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Women and Steroids

It’s no secret, anabolic steroids are widely-used by a huge part of our society; in the U. S. alone more than 6 million adults supplement with anabolic steroids but usually shocking to many people is how many females on anabolic steroids make up a portion of this overall amount. It is true, males make up the largest part of the group, in-truth it’s not just a close comparison but there are several women on steroids also and several of these women using anabolic steroids make amazing results.

Although there are many women on anabolic steroids to ensure most to have good results in-terms of both overall performance while keeping femininity certain factors must necessarily be realized. Anabolic steroids can cause significant virilization; this simply means the usage of roids may bring on masculine characteristics and other feminine related side-effects such as and not limited to:

Deepening of the voice, body hair and clitoral growth.

For the majority of women the key to success is keeping away from highly androgenic steroids like testosterone, Trenbolone, Methyltestosterone, Anadrol, Dianabol.. Most women prefer less androgenic more anabolic roids such as Anavar, Winstrol and Primobolan are often very ideal for the female athlete as they can be utilized safely and efficiently. Even so, this doesn’t mean the various ladies on steroids are not using other anabolic steroids; in-fact many women on anabolic steroids are using roids in a very similar manner as men. For instance, use of the male hormone testosterone can have serious outcomes; however, low doses of testosterone can be very beneficial in-terms of both performance and sex drive for women but most will always keep the dosage very low.

We recommend women who want to use anabolic steroids to exceed their natural potential to use mild steroids; Anavar, Primobolan and Winny for short periods no longer than 6-8 weeks. They should alwys keep the dosages low like 10-20 mg daily for anavar and 100-200 mg weekly injections of Primo depot. If virilization effects appears you should discontinue.

Most important is responsible use and that is personal education; like guys, females on steroids are advised to educate themselves on the subject as self-education will cause a larger more beneficial experience.

Anabolic steroids: Mechanism of Action and Effects on Performance

Anabolic steroids are drugs that resemble androgenic hormones (sometimes called male hormones) such as testosterone. Athletes consume them in the hope of gaining weight, strength, power, speed, endurance, and aggressiveness. They are widely used by athletes involved in such sports as track and field (mostly the throwing events), weight lifting, and American football. However, in spite of their tremendous popularity, their effectiveness is controversial. The research literature is divided on whether anabolic steroids enhance physical performance. Yet, almost all athletes who consume these substances acclaim their beneficial effects. Many athletes feel that they would not have been as successful without them.

There are several possible reasons for the large differences between experimental findings and empirical observations. An incredible mystique has arisen around these substances, providing fertile ground for the placebo effect. The use of anabolic steroids in the “real world” is considerably different from that in rigidly controlled, double-blind experiments (in a double blind study, neither the subject nor experimenter knows who is taking the drug). Most studies have not used the same drug dosage used by athletes. Institutional safeguards prohibit administration of high dosages of possibly dangerous substances to human subjects. Subjects in research experiments seldom resemble accomplished weight-trained athletes. Under these conditions, we must assess the results of sound research studies, as well as clinical and empirical field observations, in order to obtain a realistic profile of the use, effects on performance, and side effects of these substances.

How Anabolic Steroids Work

Male hormones, principally testosterone, are partially responsible for the tremendous developmental changes that occur during puberty and adolescence. Male hormones have androgenic and anabolic effects. Androgenic effects are changes in primary and secondary sexual characteristics. These include enlargement of the penis and testes, voice changes, hair growth on the face, axilla, and genital areas, and increased aggressiveness. The anabolic effects of androgens include accelerated growth of muscle, bone, and red blood cells, and enhanced neural conduction.

Anabolic steroids have been manufactured to enhance the anabolic properties (tissue building) of the androgens and minimize the androgenic (sex-linked) properties. However, no steroid has eliminated the androgenic effects because the so-called androgenic effects are really anabolic effects in sex-linked tissues. The effects of male hormones on accessory sex glands, genital hair growth, and oiliness of the skin are anabolic processes in those tissues. The steroids with the most potent anabolic effect are also those with the greatest androgenic effect.

Steroid Receptors

Steroid hormones work by stimulation of receptor molecules in muscle cells, which activate specific genes to produce proteins. They also affect the activation rate of enzyme systems involved in protein metabolism, thus enhancing protein synthesis and inhibiting protein degradation (called an anti-catabolic effect).

Heavy resistance training seems to be necessary for anabolic steroids to exert any beneficial effect on physical performance. Most research studies that have demonstrated improved performance with anabolic steroids used experienced weight lifters who were capable of training with heavier weights and producing relatively greater muscle tension during exercise than novice subjects. The effectiveness of anabolic steroids is dependent upon unbound receptor sites in muscle. Intense strength training may increase the number of unbound receptor sites. This would increase the effectiveness of anabolic steroids.

Anti-Catabolic Effects Of Anabolic Steroids

Many athletes have said that anabolic steroids help them train harder and recover faster. They also said that they had difficulty making progress (or even holding onto the gains) when they were off the drugs. Anabolic steroids may have an anti-catabolic effect. This means that the drugs may prevent muscle catabolism that often accompanies intense exercise training. Presently, this hypothesis has not been fully proven.

Anabolic steroids may block the effects of hormones such as cortisol involved in tissue breakdown during and after exercise. Anabolic steroids may prevent tissue from breaking down following of an intense work-out. This would speed recovery. Cortisol and related hormones, secreted by the adrenal cortex, also has receptor sites within skeletal muscle cells. Cortisol causes protein breakdown and is secreted during exercise to enhance the use of proteins for fuel and to suppress inflammation that accompanies tissue injury.

Anabolic steroids may block the binding of cortisol to its receptor sites, which would prevent muscle breakdown and enhances recovery. While this is beneficial while the athlete is taking the drug, the effect backfires when he stops taking it. Hormonal adaptations occur in response to the abnormal amount of male hormone present in the athlete’s body. Cortisol receptor sites and cortisol secretion from the adrenal cortex increase.

Anabolic steroid use decreases testosterone secretion. People who stop taking steroids are also hampered with less male hormone than usual during the “off” periods. The catabolic effects of cortisol are enhanced when the athlete stops taking the drugs and strength and muscle size are lost at a rapid rate.

The rebound effect of cortisol and its receptors presents people who use anabolic steroids with several serious problems:

  1. Psychological addiction is more probable because they become dependent on the drugs. This is because they tend to lose strength and size rapidly when off steroids. To stave off deconditioning, athletes may want to take the drugs for long periods of time to prevent falling behind. 
  2. Long-term administration increases the chance of serious side-effects. 
  3. Cortisol suppresses the immune system.

This makes steroid users more prone to diseases, such as cold and flu, during the period immediately following steroid administration.

Psychological Effects

Some researchers have speculated that the real effect of anabolic steroids is the creation of a “psychosomatic state” characterized by sensations of well being, euphoria, increased aggressiveness and tolerance to stress, allowing the athlete to train harder. Such a psychosomatic state would be more beneficial to experienced weight lifters who have developed the motor skills to exert maximal force during strength training. Diets high in protein and calories may also be important in maximizing the effectiveness of anabolic steroids.

Anabolic Steroids and Performance

The effects of anabolic steroids on physical performance are unclear. Well controlled, double blind studies have rendered conflicting results. In studies showing beneficial effects, body weight increased by an average of about four pounds, lean body weight by about six pounds (fat loss accounts for the discrepancy between gains in lean mass and body weight), bench press increased by about 15 pounds, and squats by about 30 pounds (these values represent the average gains for all studies showing a beneficial effect). Almost all studies have failed to demonstrate a beneficial effect on maximal oxygen consumption or endurance capacity. Anabolic steroid studies have typically lasted six to eight weeks and have usually used relatively untrained subjects.

Most changes in strength during the early part of training are neural: increased strength is mainly due to an improved ability to recruit motor units. Anabolic steroids affect processes associated with protein synthesis in muscle. Studies lasting six weeks (typical study length) would largely reflect neural changes and could easily miss the cellular effects of the drugs.

The gains made by athletes in uncontrolled observations have been much more impressive. Weight gains of thirty or forty pounds, coupled with thirty percent increases in strength, are not unusual. Such case studies lack credibility because of the absence of scientific controls. However, it would be foolish to completely disregard such observations because the “subjects” have been highly trained and motivated athletes.Please see the articles on pharmacology of sport and sports medicine in the countries of the former Soviet Union for more information on anabolic steroids.

Anabolic Steroids and Male Pattern Baldness

Over the last 10+ years I have been consulting with prospective steroid users, there is one side effect which consistently seems to concern more would-be users than anything else. This is the fear of hair loss. While some individuals don’t seem to be bothered by this, the overwhelming majority view this side effect in an extremely negative light. For many, the belief that steroid use is synonymous with hair loss is the overriding, although largely ignorant factor influencing the decision to abstain. In truth, there are numerous factors which can potentially play a role in the hair loss process, some of which have nothing to do with drug use.

Fortunately, many of these factors can either be controlled or eliminated altogether. However, in order to understand how steroid use can affect one’s hair line, we will first need to look at the genetic component involved and how anabolic steroids can accelerate this inherited trait.

The most common form of baldness in men is Male Pattern Baldness; also known as androgenic alopecia. Men affected by this condition lose their hair in a well- defined pattern starting on the sides of the temples and gradually receding from the front of the forehead back towards the crown. While Male Pattern Baldness is also associated with variations to this pattern, significant uniformity will still be present. In short, androgenic alopecia is an inherited sensitivity to the presence of androgens in the scalp, particularly the hair follicles. When these hair follicles are repeatedly subjected to the affects of androgens over time, they shrink, resulting in a thinning of the hair. Eventually, the hair follicle will close up completely, preventing any further growth.

Male Pattern Baldness is partially determined by a genetic variant in the androgen receptor on the X chromosome. Males have one X chromosome and one Y chromosome, whereas females have two X chromosomes. Since males always inherit their X chromosomes from their mothers, the genetic variant found in the AR (androgen receptor) responsible for causing Male Pattern Baldness, is inherited maternally. Still, this does not tell us the whole story, as science is now confirming that fathers are also capable of passing on genes associated with Male Pattern Baldness, particularly a variant located on Chromosome 20. Regardless of paternal-maternal liability, it is clear that there is a strong genetic component involved in hair loss and anabolic steroids have the ability to substantially speed up this process.

Does this mean that anyone who is genetically predisposed to Male Pattern Baldness cannot use steroids or else they must suffer the consequences or premature hair loss? NO, in most cases, it does not (although selection may be limited or ancillary drugs necessary). As mentioned previously, there are multiple factors which play a role in the hair loss process and if one wishes to minimize/avoid this side effect as much as possible, then applying a combination of scientific research, anecdotal evidence, and examining personal response will play key roles.

For those who carry the Male Pattern Baldness trait, administering the hormone testosterone at supra-physiological levels (unless accompanied by a 5 alpha-reductase inhibitor, which we will get to in a minute) is an off-limits practice. The reason for this is simple. Testosterone converts into a metabolite in the body known as DHT (Dihydrotestosterone) through the 5 alpha-reductase enzyme and it is this metabolite which is responsible for attaching to the androgen receptor at the hair follicle and initiating hair loss. By administering excess testosterone, the user will only increase the amount of hormone this enzyme has to work with, increasing the rate of DHT conversion and hastening the hair loss process. There is significant variance in the rate of hair loss among those who administer supra-physiological doses of testosterone, while carrying the MPB gene. More importantly, the individual cannot always know whether he carries this gene or not (unless he begins to experience Male Pattern Baldness), nor can he know how quickly he will lose his hair should he decide to use testosterone.

In those who are not sensitive to the effects of DHT on the hair line, testosterone use, even when used at extremely high dosages, will not cause hair loss. We see this demonstrated in the lives of professional bodybuilders all the time. It is safe to say that all of today’s professional bodybuilders use significant dosages of testosterone, with most relying on testosterone as their base drug. However, it is quite normal to see some pro BB’rs with a full head of hair after many years of high-dose testosterone use, while other pro bodybuilders will be nearly completely bald by age 30 (or sooner).

For those men who consider hair maintenance a priority, but who still want to experience the benefits anabolic steroids have to offer, I recommend low-dose testosterone use while “on-cycle”. The reason I recommend low-dose use and not its complete elimination from one’s drug repertoire when running a “cycle” is because the body is reliant on testosterone for proper male functioning, including sexual function. By using steroids in the absence of testosterone, the body will eventually shut-down its own natural testosterone production, leading to a cascade of unpleasant side effects. By administering low-dose testosterone in conjunction with other steroids, DHT conversion will remain minimal, keeping hair loss at bay, while maintaining normal physiological functioning (Note: Certain steroids can cause sexual dysfunction even in the presence of testosterone, but that is another topic for another day).

DHT is not the only mechanism potentially responsible for steroid related hair loss. For example, some individuals may be able to get away with administering large dosages of testosterone, but as soon as they begin utilizing other non-DHT converting steroids, they run into problems. Clearly, in cases such as this, DHT is not the cause. Another potential contributor to hair loss is the androgenic potency of a steroid. While this factor is not always relevant, the majority of potent androgens are more likely to result in hair loss compared to steroids characterized by a weaker androgen component.

There is a myth being spread around both the net and the magazines today regarding which steroids are more likely to cause hair loss, but before we get to that, we must first gain an understanding of the subject of steroid “families”. Every anabolic steroid sold on the market today technically belongs to one of three classes (or families) of steroids. These are the testosterone-based, 19 nor-based, and DHT-based families of steroids. Each anabolic steroid is defined as belonging to one of these three classes of steroids, based on which of the three molecules it was originally derived from. For example, since the steroid Anavar is an alteration of the DHT molecule, it is classified as belonging o the DHT family of steroids. Since Dianabol is an alteration of the testosterone molecule, it is classified as belonging to the testosterone family of steroids. In reality, “all” steroids are derived from the testosterone molecule itself, but we utilize these 3 classes of anabolic steroids as basic sub-categories, in order to further categorize steroids into more specific groups.

With that said, it has often been claimed that those anabolic steroids which belong to the DHT family of steroids are more likely to cause hair loss simply because they were originally developed from the DHT molecule. This is blatantly untrue. There are many DHT-based steroids which have been heavily used by both BB’r and other athletes and have been anecdotally proven, through decades of user experience, to be much less likely to cause hair loss compared to numerous testosterone and 19-nor based steroids. As an example, the steroids Superdrol and Anavar both belong to the DHT class of anabolic steroids, yet these steroids have become well-known as some of the least likely to result in hair loss. This is one of the primary reasons why Anavar has long been considered the go-to steroid for 1st time womenanabolic steroids users, as it is one of the least likely to initiate masculinizing side effects, including hair loss. In contrast, the 19-nor based drug, Trenbolone, and the testosterone-based drug, Halotestin, are both much more likely to cause hair loss in the average user, despite their non-DHT based status!

This brings me to an important point. When determining which steroids are generally more likely to cause hair loss, we should NEVER look to a steroid’s “family” as a causative factor. Rather, each steroid must be evaluated on a case by case basis. We cannot clump steroids into groups in an attempt to accurately pinpoint which drugs are the prime offenders. It just doesn’t work that way. Class is completely irrelevant as proven through the collective experience of generations of bodybuilders and anyone who is knowledgeable regarding how steroids work in the real-world knows better than to make such a sweeping generalization.

When it comes to evaluating each steroid on a case by case basis, real-world experience may have shown us which steroids are the most likely to bring about thinning of the hair, but there is one other important factor we cannot ignore when it comes to hair loss. This is called personal response. It is important to remember that as individuals, none of us respond identically to all the various bodybuilding drugs. Steroids have many different effects on the human body and each steroid affects the body in its own unique way. There are some effects which we all experience when using certain steroids (although to what extent may vary), but there are other effects which only apply to the majority. Every now and then, an individual will break away from the norm and respond in a different manner than the majority. It is no different when it comes to hair loss. We may know which steroids are the most and least “likely” to cause hair loss based on the experience of generations of 
users, but this does NOT mean “everyone” will always have the same experience, as personal response can and does vary. For this reason, only recommendations can be given…not guarantees.

Below I list a few of the most commonly known steroids, which have been proven to be among the least friendly on the hair line in the majority of users:

  • Testosterone
  • Trenbolone
  • Halotestin
  • Proviron
  • Methyltestosterone
  • Anadrol
  • Dianabol
  • Masteron

Below I list a few of the most commonly known steroids, which have been proven to be among the most friendly on the hair line in the majority of users:

  • Anavar
  • Nandrolone
  • Boldenone (low-moderate risk)
  • Turinabol
  • Primobolan
  • Testosterone used with an appropriately dosed 5-AR inhibitor

Bodybuilding Antagonistic Muscles Workout Split

In an antagonistic muscles workout split, bodybuilding workouts are designed to train body parts for opposing muscle groups each day. This is one of my favorite ways to train in the off-season and was one of the ways in which bodybuilding legends like Dave Draper and now Governor Arnold Schwarzegger used to train most of the time.

There are several advantages to this workout split:

  1. It can save time if you decide to superset opposing muscle groups such as the chest and the back.
  2. It can help you gain strength if you decide to alternate between sets of one muscle group (such as the chest) and sets of the opposing muscle group (such as the back) while you rest in between.

For instance, if you perform an incline bench press followed by 90 seconds of rest, and then perform a wide grip pull-up, followed by 90 seconds of rest, you will be resting a total of three minutes plus the time it took to perform the pull-ups before you go back to the incline bench again. You will also notice that your strength for each exercise sometimes increases as the nervous system seems to have an easier time recovering between sets when this technique is used.
There are a couple of ways in which I have set up an antagonistic mucle group bodybuilding workout. You can use a three day split or a four day split:

Three Day Split

In this split, the whole body is worked over a period of three days:

Day 1 - Chest/Back/Abs

Day 2 - Thighs/Hamstrings/Calves

Day 3 - Shoulders/Biceps/Triceps

Training Notes

As a good rule of thumb, you can perform 10-12 sets for the big body parts (Chest, Back, Thighs, Hamstrings, Shoulders) and 8-10 sets for the little ones (Abs, Calves, Biceps, Triceps). More genetically gifted bodybuilders may be able to get away with more, but this amount works for most.

Frequency wise, three days on and one day off is good for mass gains.

If you are looking for fat loss, doing six days in a row and then resting on the seventh works best if time allows. You can also rotate days doing 5 days on and two days off (which leaves the weekends off), or three days on, one day off, 2 days on, and one day off (which allows for Thursday and Sundays off).

Hardgainers benefit most from doing two days on, one day off, one day on, and one day off. Alternatively, they can also do just workout Monday, Wednesdays and Fridays, doing each workout just once per week.

Four Day Split

In this split, the whole body is worked over a period of four days:

Day 1 - Chest/Back

Day 2 - Thighs/Hamstrings

Day 3 - Shoulders/Calves

Day 4 - Biceps/Triceps/Abs

Training Notes

Because this workout splits the body over four days, you can do more sets for each muscle group. This split is better suited for a more advanced bodyuilder. As a good rule of thumb, you can perform 12-15 sets for the big body parts (Chest, Back, Thighs, Hamstrings, Shoulders) and 12 sets for the little ones (Abs, Calves, Biceps, Triceps). More genetically gifted bodybuilders may be able to get away with more, but this amount works for most.

Frequency wise, four days on and one day off is good for mass gains.

If you are looking for fat loss, four days on and one day off works well also.

Hardgainers benefit most from doing two days on, one day off, two days on, and two day off.

Real Results Beastly Basics Fullbody Workout

This fullbody workout focuses on the basics, while utilizing minimal isolation work. You will hammer the body with effective compounds lifts that will stimulate plenty of muscle growth, and you will add strength rather quickly.

This program works well for lifters who train alone, or at home, and must constantly shuffle and strip weight from barbells and dumbbells. The added downtime from changing weight can lengthen a workout. With the Beastly Basics approach, you will use only 5 lifts per session, keeping workouts to approximately 60-70 minutes each day.

Perform this workout on Monday, Wednesday and Friday.

Workout Notes

Rows. Rows can be either:
Barbell Rows – 5×5
Dumbbell Rows – 3×10

For dumbbell rows, warmup as required. I find that dumbbell rows seem to work better with slightly higher reps, so resist the urge to use a 5×5.
Curls. You may use any dumbbell curling variation you prefer.
5×5. 5×5 protocols incorporate 2 ramped sets. The percentages of these working warmup sets are based on the working weight used during the final 3 sets.

Set 1 – 60% x 5 reps
Set 2 – 80% x 5 reps
Sets 3-5 – 100% x 5 reps

4×8.

The 4×8 sets will involve a slightly lighter weight when compared to Monday’s workout. The first 2 sets are ramped.

Set 1 – 60% x 8 reps

Set 2 – 80% x 8 reps

Sets 3-4 – 100% x 8 reps

Use this same ramping structure for seated overhead dumbbell presses on Friday.

Seated Dumbbell Presses. You may use either the double arm version, or perform them one arm at a time. As the weight gets heavier, it may be easier to use the one arm version. Swinging two heavy dumbbells into place can be a cumbersome task.

Pull Ups. Make sure you are performing pull ups (palms facing away) and not chin ups. If you can’t perform any pull ups, substitute in dumbbell rows or seated cable rows, and perform 3 sets of 10 reps.

Weighted Sit Ups. Hold a 10, 25 or 45 pound plate on your chest. Perform up to 25 reps per set. While weighted sit ups are a very effective choice, you can really use any abdominal exercise that allows progressive resistance such as rope cable crunches.

Calf Raises. These are performed standing while holding a barbell. You may choose to perform calf raises with the balls of your feet upon a 5 or 10 pound plate, but this is not a requirement. You may also use a standing or seated calf raise, or calf raises on a leg press machine.

Dips. If 3 sets of dips become too easy, meaning you can perform 10 dips on each set without effort, add 2 more sets. You may also want to consider either adding resistance via a dipping belt and keeping the set total to 3.

Progression – Adding Weight

Important Note: Never train to failure on any set. Stop each set when you feel like you may not be able to complete the next rep, or stop a set when you feel your exercise form is becoming sub-par. Performing sloppy reps leads to injury. It’s ok if you can’t make the rep goal/target for each set. Try to get it the next time!

Your goal each workout is “beat” your previous performance on a given exercise by at least one rep. This will not always happen. Expect at least one to two unproductive workouts each month.

For exercises that utilize the 5×5 structure, you can add weight in a number of ways:

  • Weekly. If you are an experienced beginning lifter, you can attempt to add 5 pounds each week.
  • Bi-Weekly. Adding 5 pounds every other week is also a great way to approach progression. Over the course of a year you will have the potential to add 120 pounds to a lift.
  • Monthly. For intermediate lifters who will not experienced rapid strength gains, try adding 5-10 pounds per month.

At some point you will be unable to hit 5 reps for the last 3 sets. At this point you will need to make some adjustments. If after a couple weeks you are unable to perform a 5×5 with a given weight, you will need to follow some advanced progressional approaches.

All other exercises. For these exercises, when you can perform the top level number of reps for all of the sets, add weight the next time you perform this exercise. It is wise to add only 5 pounds.

Trenbolone Cycle Information.Trenbolone Acetate / Trenbolone Enanthate

Trenbolone is a very powerful steroid that has never been FDA approved for use in humans. It was originally developed as finaplex pellets for use as a vetinary product to be put under the skin of cattle. However over time bodybuilders have realised its unique properties and powerful benefits and it has become a favourite anabolic steroid for many, despite having more harsh side effects than most other steroids. Many people would convert the pellets into an injectable form, in a rather crude and dangerous manner that would neither be safe nor sterile, and poses many risks. However, despite being non-approved by the FDA, there are a number of respectable Underground Labs (UGLs) that produce quality injectable forms of Trenbolone. For the user who understandably likes to stick to pharmaceutical grade steroids, unfortunately there is no such form of Trenbolone available.

Trenbolone is a highly androgenic steroid, with binding to the Androgen Receptor (AR) in the region of three times as high as testosterone. It does not aromatise and so is not subject to estrogenic side effects. In addition to high androgenicity, it is also extremely anabolic too, thus is very good at building muscle mass, and retaining muscle mass in a calorie deficient mode. It is also thought that Trenbolone inhibits cortisol production directly through the glucocorticoid receptors. Trenbolone is often found to be a body transforming drug, and also can aid a little in fat loss. This may be due to the very strong binding of Trenbolone to the AR, which has been postulated to be one mechanism that results in the activation of fat loss pathways, possible through direct binding to fat cells’ ARs. This makes Trenbolone a favourite among bodybuilders for cutting, and in addition to these benefits, trenbolone usually results in large increases in strength due to its high androgenic effects.

Trenbolone although not converted to estrogen, does have progesteronic effects, which will be discussed further in the side effects section of this article.

Typically today underground labs produce Trenbolone acetate as 75g/ml or 100mg/ml. It is often recommended first-time users of Trenbolone to use the faster acting acetate in case the side effects become too much for the user, they can then come off of the steroid very quickly and it is out of the system much quicker than, for example, the enanthate ester. For the novice user, 75mg or 100mg every other day (eod) is advised, however due to the acetate ester being even shorter than a propionate ester and the half life 1 day or less, to both reduce sides and aid gains, it is advisable that the user (if they can bear every day injections) injects trenbolone acetate every day (ed), at 37.5-50mg ed.

More advanced users may find that taking the Trenbolone to amounts over 500mg per week has very desirable effects on strength and body composition, however note that the side effects will also increase with the increase in dose. Due to the negative effect that Trenbolone has on libido, it is not generally recommended to take Trenbolone without testosterone. However, one can take Trenbolone for short periods without testosterone and introduce an aid such as Proviron (mesterolone) to help with the libido issues, along with proper extensive post cycle therapy (PCT) for recovery. A typical test-free cycle with Trenbolone may include something like 600mg Primobolan per week, 400mg Trenbolone Enanthate per week, for 10 weeks, PCT starting 2 weeks after last injections. The enanthate ester and other similar esters of Trenbolone can be injected twice per week. Below are some example cycles using trenbolone:

Novice:

Testosterone propionate 100-150mg eod, 6-8weeks
Trenbolone acetate 75-100mg eod, 6-8 weeks, PCT 4 days after last prop injection.

Intermediate:

Testosterone enanthate 750mg per week, weeks 1-12
Trenbolone enanthate 400mg per week, weeks 1-12
Winstrol 50mg ed weeks, 8-14

Primobolan 600mg per week, weeks 1-10
Testosterone propionate 200mg eod weeks 1-12
Trenbolone enanthate 400mg per week, weeks 1-10

Advanced:

Primobolan 600mg per week, weeks 1-10
Trenbolone enanthate 400mg per week, weeks 1-10
Testosterone enanthate 1000mg per week, weeks 1-12
Trenbolone enanthate 500-700mg per week, weeks 1-12
Anavar 80-100mg ed, weeks 1-14

Very advanced/pre-contest:

Testosterone propionate 100-200mg ed
Trenbolone acetate 75-100mg ed
Masteron 400-600mg per week
Winstrol 50mg ed
Primobolan 600mg per week
Halotestin 10-20mg ed

Out of all the injectable steroids available, Trenbolone is the one that should be used with extreme caution and only after plenty of research into its side effects and common cycles have been carried out. Trenbolone side effects can be very bad to many users, so much so that they will not use it despite its very positive effects on the body and strength. Firstly, as Trenbolone is so androgenic, all side effects that are seen with strong androgens can be expected (if prone) with Trenbolone. If one is prone to male pattern baldness (MPB) than Trenbolone will likely speed this up. Some users find acne on trenbolone worse than when on any other steroid. Certainly Trenbolone is not recommended for female users due to its strong androgenic properties and the common side effects that manifest themselves in females who use strong androgens.

Despite the fact that Trenbolone cannot aromatise, due to the progesterone route it can cause things like gynecomastia, but this will only really happen in the presence of estrogen. This does happen though in many users, as Trenbolone is usually stacked with a testosterone, which obviously can and will convert to estrogen. Gynecomastia from Trenbolone can be quite bad many will find, however if you do not suffer from this than other estrogenic side effects should not be of worry, as Trenbolone does not cause any water retention or similar, but in fact often gives a hardened look and feel to the muscles.

Trenbolone also seems to give many users poor sleep patterns and insomnia. In addition, it can cause severe sweating in many, both during the night time and also just from doing the smallest of activities such as walking up stairs, etc. It also can impair to a certain degree, cardiovascular function, which means that it is not ideal for use in those who regular partake in such sports or activity that require a decent level of cardiovascular fitness.

Trenbolone also increases blood pressure in many users, some to such a degree that they have to cease using it. Thus it is recommended that one who wishes to use trenbolone, invests in a blood pressure monitor so they can regularly measure their blood pressure and keep an eye on it throughout the cycle.

Many people claim that Trenbolone has a negative effect on the kidneys. There are many of these claims certainly across the Internet since its use has become more widespread. However, there is no real evidence for these claims, and certainly I have seen many long-term users of trenbolone have kidney function tests that are well within the normal range. Perhaps the reason for this theory is the fact that when using Trenbolone, many find that their urine can become a much darker more orange-brown colour. However, this is due to the fact that Trenbolone undergoes very little modification or breakdown and is excreted as a rust-coloured oxidised form in the urine. In addition to this, any damage to kidney may not even be directly due to the Trenbolone, but more to do with the increased sweating and water loss from excessive body heat whilst on Trenbolone, without the sufficient addition of water intake. Thus it is recommended if running Trenbolone to keep the water intake high.

As Trenbolone is such a strong steroid, it is very harsh on the HTPA axis and will shut down the body’s natural testosterone production very easily and, for many, very harshly. It is comparable to ‘deca dick’ that people can experience with deca, and longer cycles may need to include the use of HCG to restore one’s own natural production of testosterone. Recovery from cycles containing Trenbolone is not easy, and requires a very well thought out and stringent PCT routine and diet.

It has also been suggested through research that Trenbolone actually (although aiding slightly in fat loss) reduces endogenous T3 levels. Thus some advocate the use of 25mcg T3 throughout a Trenbolone cycle. This writer does not personally think that this is necessary; however it is something that users may wish to consider when using Trenbolone, especially if their natural T3 production is on the lower side of the normal range. It is a very good idea to get blood work done both before and after any cycle including Trenbolone.

Tren cough

The so called ‘tren cough’ or ‘Fina cough’ is well known amongst many tren users. Some users seem to get the cough following every injection; others never or extremely rarely will get the cough. Usually it is manifested upon injection, with a tightness in the chest, and a metallic taste in the back of the mouth, followed by an uncontrollable violent cough which can be quite frightening, as anyone who has experienced it will tell you, whether it’s for the first time or not. There have been some very elaborate theories about the reasons for getting the cough from Trenbolone, some of which have had mechanisms involving molecules that only Trenbolone affects resulting in bronchioconstriction, etc. However, the fact remains that many users have also experienced the same cough from steroids such as Equipoise and Testosterone cypionate. In addition, these mechanisms that are proposed are highly unlikely to occur immediately upon injection, as that is too fast a timescale for the proposed mechanism. Thus it must be the result of something entering the blood stream and traveling to the lungs for the cough to be manifested that quickly.

This leads us onto the next theory suggested by many which is that trenbolone is produced by many UGLs, and as such is made with higher percentages of Benzyl Alcohol (BA) than pharma grade products are, and it is the alcohol that is causing the reaction. The only problem with this theory is that Trenbolone is made by most UGLs with the same BA percentages as things such as Testosterone propionate, and Nandrolone decanoate. If it was purely the BA concentration, than we would expect to see the cough with these other products as well, which we do not. Thus, as we have eliminated the oil, solvents and carriers, it leaves us with the Trenbolone product itself as the potential culprit.

One thing that you notice about Trenbolone is that it is often a golden-brown / rust colour when in oil solution. If the hormone powder is refined to greater than 99.5% purity or so, then the colour of Trenbolone in solution actually gives a very light golden colour, much like other testosterone products; however, refining the hormone to this level of purity is extremely difficult. This is why there is colour variation from batch to batch with different underground labs; something as small as 0.1% purity can affect the colour of the final product.

As mentioned above when discussing kidney effects of Trenbolone, the oxidised Trenbolone is a rust colour – much like the colour seen of trenbolone in oil solution. What you also notice with steroids such as Equipoise and to a lesser degree, Testosterone cypionate, is that these steroids too are hard to very highly refine and often a browny-rust colour, more so than products such as Testosterone propionate, etc. It is very likely then that these oxidised particles get into the blood stream upon injection and this causes some sort of anaphylactic (allergic) reaction in the lungs as the particles react with the alveoli, perhaps. This seems to be confirmed by the fact that the darker the Trenbolone is the more likely one is to get a cough (personal and general experience). The best way to try and avoid this is to firstly inject very slowly and not move the needle around after aspirating, and also mixing the Trenbolone with another product such as test prop.

Trenbolone Cycle

A good Trenbolone cycle can be one of the best cycles any performance enhancer will ever complete and while there are many forms and plans to choose from disappointment is rarely a concern you will have. As it is the easiest to control and the most widely available Trenbolone Acetate will be our preferred and recommended Tren form; it is not a must, you can use Trenbolone-Enanthate or Trenbolone- Hexahydrobencylcarbonate (Parabolan) if you so choose but the Acetate form is generally the most efficient. As you may already understand a Trenbolone cycle can be during a period of growth or dieting and in most cases it is equally effective in both; in-fact, it is such an amazing steroid many performance enhancers choose to include it in most of their cycles regardless of purpose.

The Basic Trenbolone Cycle:

While it may be a basic Trenbolone cycle that by no means implies that it is weak but this will be a good place for a first time Tren user to start and in many cases it will be as extensive as many will ever need to go. Your basic Trenbolone cycle will always include testosterone; in-fact we must recommend testosterone with all Trenbolone cycles as the Tren hormone is very suppressive to natural testosterone production; so much so that without testosterone therapy you would have no testosterone in play.

For this Trenbolone cycle our dosing will be at 50mg every other day; if you tolerate that dosing well and still want more 75mg every other day can be well justified. In most cases, regardless of the specific dose the total duration of use will be for 8 weeks; 12 weeks can be used but 8 is far more tolerable by most and in the name of health generally a good place to end. If you are going to enter the 12 week mark of use it is generally better to have some experience with the hormone and to be practicing such methods for very specific purposes such as a competition. For the off-season Trenbolone cycle you will find it stacks very well with testosterone and Dianabol as well as Anadrol if you so choose. During your cutting phase Tren will stack well again with testosterone and other anabolics such as Winstrol and Anavar.

Advanced Trenbolone Cycle:

An advanced Trenbolone cycle will always begin at 100mg every other day and often and in many cases creep its way into dosing levels of 100mg every single day during use. Such every day doses are generally not needed for the off-season athlete; such doses are not going to create enough added growth to warrant the added physical stress; however, daily doses of 100mg per day can be very useful during a diet, particularly later on in the diet close to a competition.

For the off-season advanced Trenbolone cycle of 100mg every other day many will find a combo of Test/Tren/Dbol to be just about unbeatable but for the truly advanced users we at many times recommend an overlapping method. For an effective overlapping Trenbolone cycle the individual might supplement with testosterone and Deca-Durabolin for 12 weeks but at the 10 week mark he would have added in Tren to the equation and by week 12 discontinued his Deca and stuck with Tren and Testosterone only. An overlapping Trenbolone cycle might look like this (example does not include orals such as Dianabol or Anadrol):

  1. Week 1-20 Testosterone
  2. Week 1-12 Deca-Durabolin
  3. Week 10-20 Trenbolone 

For the dieting or cutting Trenbolone cycle this is where things can get really exciting as the choices are truly endless. In most cases you will be best served by supplementing with your Tren the latter half of the dieting phase; for example, if you are going to diet for 16 weeks you will want to use the Tren the last 8-12 weeks rather than from the start. You can also use the same overlapping method here as well; many athletes enjoy a Trenbolone cycle that overlaps with Equipoise during this period of use. As for other items, Winstrol, as always testosterone, good AIs and fat burners and if you can swing it a good batch of HGH and youll have a cycle that’s hard to beat.

Trenbolone (Acetate / Enanthate). Tren Steroid Cycles and Dosages

Trenbolone is a very powerful steroid that was originally manufactured as pellets that were administrated beneath cattle skin, later to be brewed by underground labs for use by anabolic steroid users. Trenbolone has never been approved by the FDA for use in humans, and the drug is commercially used as vetinary finaplex pellets. Trenbolone is now widely available via many underground labs, and is now a very popular steroid due to its very strong and potent effects.

A highly anabolic and androgenic steroid, Trenbolone can be a very harsh steroid for users that are prone to androgenic related side effects, and even those who have previously not been. Trenbolone has been said to bind to the androgen receptor at a rate as highly as three times that of testosterone, so we can strongly expect androgenic related side effects to result from its use in users that are prone to such side effects. Acne can be expected in prone users, and those who often have a break out of acne from use of other steroids, such as testosterone products, may wish to think about running a different compound. Accutane is sometimes taken with Trenbolone to combat the acne. Trenbolone will almost certainly speed up male pattern baldness (MPB) due to its very strong androgenic effects, and therefore will not be a suitable steroid for those who are prone to MPB and wish to avoid it.

Sweating and a decrease in cardiovascular capability is often widely reported with Trenbolone usage. Trenbolone is not really suitable for those who participate in sporting activities that require cardiovascular fitness. Sweating is also reported greatly at night time, with insomnia and strange dreams. Water intake should be efficient when taking Trenbolone due to water loss.

Although Trenbolone does not aromatise, in the present of oestrogen Trenbolone can manifest progesterone related side effects, such as gynecomastia. This should be kept in mind when stacking with other androgenic anabolic steroids, such as testosterone. Trenbolone is also very harsh on the HTPA function, and recovery is often said to be harsher compared to other steroids. The use of HCG is often advised for this reason.

Trenbolone (Tren) cough is a widely experience side effect from Trenbolone usage, but the cough has also been reported in the use of other anabolic steroids. The cough itself is not a pleasant experience, usually starting with a strange taste in the mouth followed by a strong and uncontrollable cough. What causes Tren cough is a much disputed topic in the bodybuilding community, with claims of it being caused the amount of Benzyl Alcohol used in its production by the labs, or by the oxidised partials that get into the blood stream of the user upon administration of the Trenbolone.

Users can expect rapid gains in muscle size and strength from the use of Trenbolone. The steroid is often use in cutting cycles due to its ability to cause hardening of the muscles, and positively changing a persons body composition. Trenbolone is also said to be responsible for some level of fat loss, but we should not rely on the compound for this benefit. Novice users may wish to use the faster Acetate ester due to its faster half life, and therefore the ability to cause a quicker drop in blood levels upon ceasing the drug incase any undesirable side effects become present. 75mg-100mg each other day (EOD) is common, and is often stacked with testosterone propionate. Greater dosages is sometimes administrated by more advanced users, often 500mg + per week.

Due to its high androgenic characteristics, Trenbolone is not really a suitable steroid to be taken by women.

Trenbolone is a very powerful and potent androgenic anabolic steroid produced by many respected underground labs. For those who can handle the harsh side effects it can be a very effective drug, possessing the ability to drastically change a users body composition.

Trenbolone anabolic steroid cycles

Novice user’s cycle

  • 75-100mg of Trenbolone each other day (EOD) for 8 weeks, stacked with Testosterone Propionate 100mg each other day (EOD). Start pct 4 days after last injection

Intermediate/Advanced user’s cycle

  • 500mg of Trenbolone stacked with 500mg-1000mg of Testosterone Enanthate per week for 10+ weeks.

Trenbolone is often used in competitors cuttings stacks prior to content, and stacked with many other anabolic steroids.

Post Cycle Therapy

Pulsatile secretion of gonadotropin releasing hormone (GnRH) from the hypothalamus is required for both the initiation and maintenance of the reproductive axis in the human. Pulsatile GnRH stimulates the biosynthesis of luteinizing hormone (LH) and follicle stimulating hormone (FSH) that in turn initiates endogenous testosterone production and spermatogenesis as well as systemic testosterone secretion and virilization. Failure of this episodic GnRH secretion or disruption of gonadotropin secretion results in the clinical syndrome of hypogonadotropic hypogonadism (HH).

The usage of anabolic steroids may result in a functional form of HH known as Secondary Acquired Hypogonadotropic Hypogonadism and is diagnosed in the setting of a low testosterone level and sperm count in association with low or inappropriately normal serum LH and FSH concentrations.

In order to avoid any unnecessary confusion, it is important to understand what the actions of Gonadatropin therapy and Selective Estrogen Receptor Modulators are as well as how they differ from each other and more specifically, during post cycle recovery (PCT).

Gonadotropin Therapy:

There is nothing more effective than Human Chorionic Gonadotropin (HCG). The action of HCG is identical to that of pituitary LH. This takes place independently and is not affected by exogenous hormones and/or preexisting HPTA suppression. Therefore, it directly stimulates a dramatic increase in endogenous testosterone production, spermatogenesis and testicular volume. The primary goal during the first few weeks of PCT is to quickly restore testicular volume and function. Also, the dramatic increase in testosterone production is necessary to avoid and/or minimize the unfavorable “crash” effect. In the majority of individuals with larger testes at baseline, HCG alone is sufficient in restoring endogenous testosterone production as well at the induction of spermatogenesis which is most likely a result of residual FSH secretion. Once there is a plateau in the response to HCG, treatment with an FSH preparation such as human menopausal gonadotropin (HMG) or recombinant follicle stimulating hormone (rFSH) should be added in combination to HCG.

*The addition of an FSH preparation is rarely required and is best suited for severe cases of HH. FSH preparations are not readily available to most individuals. Therefore, there is no need to go into details with respect to its application at this time.

HCG is administered by subcutaneous (SC) or intramuscular (IM) injection. The average (3ml 22-25g x ?-1½”) syringe is adequate for IM injections but insulin syringes (½-1ml 28-30g x ½-1”) are recommended for SC injections. In regards to effectiveness, there should be no discernable difference between either of the techniques. The individual should opt for the most comfortable and/or convenient form of administration.

HCG ampoules are supplied in 500, 1,000, 2,000, 5,000 and 10,000 IU preparations accompanied by 1 ml of sterile dilluent. It should be stored at a controlled room temperature (15-30 degrees C or 59-86 degrees F) and should be used immediately after reconstitution.

HCG multidose vials are supplied in 2,000, 5,000 and 10,000 IU preparations accompanied by 10 ml of bacteriostatic water. It should be stored at a controlled room temperature (15-30 degrees C or 59-86 degrees F), refrigerated (2-8 degrees C or 36-46 degrees F) after reconstitution and used within 30 days.

The terms international units (IUs) can occasionally cause confusion when reconstituting and measuring HCG. The actual process is quite elementary and the concentration per ml (cc) is dependant on the concentration of the lyophilized powder and the volume of dilluent used for reconstitution. For example, if you dilute 5,000 IUs HCG with 5ml (cc) solvent, the end result is 1,000 IUs per ml (cc). Divide the same 5,000 IUs with 10 ml (cc) and the end result is 500 IUs per ml (cc).

*Bacteriostatic water should always be utilized during reconstitution when long term (30 day) storage and multi dose administration are required.

Selective Estrogen Receptor Modulators:

Selective estrogen receptor modulators (SERMs) such as Clomiphine (Clomid) and Tamoxifen (Nolvadex) increase pituitary LH secretion in secondary manner by blocking estrogen negative feedback on the HPTA. On average, this is not strong enough by itself to counteract the severe imbalance of the androgen:estrogen ratio that is encountered post cycle, especially in the presence of testicular atrophy. Therefore, SERMs are used during PCT primarily as an anti estrogen and to continue the stimulation of pituitary LH after HCG has been discontinued.

Nolvadex is widely available in 10 mg or 20 mg tablet preparations and Clomid is available in 50 mg tablet preparations.

Before Beginning PCT:

It is highly recommended to establish baseline blood values before beginning a cycle. The same principle applies to establishing post cycle blood values, which are necessary for evaluating recovery. Post cycle blood work should be obtained approximately 4 weeks after the cessation of PCT in order to determine accurate readings. Additional blood work should be performed when applicable and/or required.

The following are Fasting blood values:

Hormone

  1. Cortisol, Total
  2. Estradiol, Extraction
  3. Prolactin
  4. LH
  5. FSH
  6. T3, Free
  7. T4, Free
  8. TSH
  9. Testosterone, Total, Free and Weakly Bound
  10. Hemoglobin A1C
  11. Fasting Insulin
  12. Somatomedian C (optional)

Cardiovascular

   13. CBC
   14. Comprehensive Metabolic Panel
   15. Lipid Panel

Other

    16. GGT Important Liver Value not included in Comp Metabolic Panel

When to begin PCT:

On average, begin PCT approximately 5-10 days after your last injection regardless of longer acting esters. Begin PCT 1-3 days after your last injection and/or intake when using short acting esters.

Keep in mind, pituitary LH secretion automatically increases as the hormones diminish from your system. The elevated androgen levels are from an exogenous source and your endogenous production is suppressed. Therefore, waiting for the exogenous androgens to completely clear from your system, ultimately results in lower total concentrations of androgens in your system when beginning PCT. This leads to an unfavorable andgrogen:estrogen ratio and the well known “crash” effect.

*As previously mentioned, the actions of HCG take place independently and is not affected by exogenous hormones and/or preexisting HPTA suppression. There are no contradictions with respect to the effectiveness of HCG usage while exogenous hormones are present in your system.

PCT Protocol(s):

1.) 1,000 IUs HCG 3x/wk (mon/wed/fri) in combination with 20 mgs Nolvadex for the first 3 weeks. After, discontinue HCG and continue with 20 mgs Nolvadex for an additional 3 weeks.

2.) 1,000 IUs HCG 3x/wk (mon/wed/fri) in combination with 20 mgs Nolvadex and 50 mgs Clomid for the first 3 weeks. After, discontinue HCG and continue with 20 mgs Nolvadex and 50 mgs Clomid for an additional 3 weeks.

3.) 1,500 IUs HCG 3x/wk (mon/wed/fri) in combination with 20 mgs Nolvadex for the first 3 weeks. After, discontinue HCG and continue 20 mgs Nolvadex for an additional 3 weeks.

4.) 1,500 IUs HCG 3x/wk (mon/wed/fri) in combination with 100 mgs Clomid and 20 mgs Nolvadex for the first 3 weeks. After, discontinue HCG and continue with 50 mgs Clomid and 20 mgs Nolvadex for an additional 3 weeks.

Option one can be considered as a standard PCT protocol. This should apply to all basic cycles. Option 2 is generally the same as option one except for the addition of Clomid which is added as a supporting recovery aid. Option three and four incorporate a higher HCG dosage and have a relationship similar to options one and two in the sense that Clomid is incorporated in the latter as a supporting recovery aid.

*The majority of my experience is with intermediate to advanced athletes whom have completed multiple cycles with higher dosages. Therefore, based upon previous blood work results and considering the common or convenient preparations available, we have established that 1,500 IUs 3x/wk (mon/wed/fri) to be the optimal HCG dosage to begin with. The Nolvadex dosage remains unchanged however Clomid is utilized throughout the entire PCT at 100 mgs during the first 3 weeks and 50 mgs for the last 3 weeks.

HCG During Cycle:

HCG in combination with Nolvadex can and should be used during prolonged (12+/wks) and high dosage (1,000+mgs/wk) cycles. In this case, 500-1,000 IUs HCG in combination with 20 mgs Nolvadex for 7-10 days consecutively is administered mid cycle or intermittently (every 6-8 weeks) during the cycle.

Maintaining testicular volume during cycle does in fact improve recovery when compared to atrophied testes when beginning PCT. This solution addresses both testicular atrophy and prevention of Leydig cell desensitization (discussed next) associated with HCG usage.

Leydig Cell Desensitization:

Leydig cell desensitization does in fact occur to some degree with prolonged or high dose HCG usage. Using it continuously during a cycle could possibly cause the LH receptor to desensitize which in turn would ultimately render the PCT to be either less effective or possibly useless. This seems counterproductive. HCG will not be needed on cycles where the proper ancillaries are used and where the dosages/durations are realistic.

The previous summary was a general statement. The reality and good news is that Leydig cell desensitization due to HCG usage is blocked and/or minimized by Nolvadex. This occurs by suppressing HCG’s ability to inhibit the conversion of 17 alpha hydroxyprogesterone to testosterone.

Additional Factors That Influence Recovery:

Factors that may complicate and/or delay recovery are elevated levels of estrogen and prolactin. Both of these hormones, when elevated, exert negative feedback on the HPTA. Estrogen and its side effects can be controlled by using an aromatase inhibitor such as Aromasin, Femara and Arimidex during cycles including aromatizing anabolic steroids. Prolactin and its side effects can be controlled by using an anti Prolactin during cycles containing nandrolones. If these measures have not been addressed during the cycle, they will more than likely need to be addressed during PCT. In this scenario, the objective is to lower these hormones to acceptable levels in order to avoid the complications and/or delay in recovery. Blood work is imperative in evaluating the effectiveness of therapy. This will provide a clear and concise answer in regards to the adjustment of dosages and continuation of medication if necessary.

*There are numerous studies which support and refute the association of nandrolones and prolactin. However, based on first hand experience and blood work results, there are far more individuals today whom can testify that the usage of nandrolones can attribute to an increase in prolactin concentrations. In addition, many individuals have reported elevated prolactin levels during cycles which do not contain nandrolones. The common factor within these cases is supraphysiological levels of estrogen. Estrogens act directly at the pituitary level by causing the stimulation of lactotrophs which in turn enhances prolactin secretion. This is another reason why estrogen management in the form of an aromatase inhibitor should be included with cycles containing aromatizing anabolic steroids. Although not absolutely necessary and considering the necessary restoration of physiological estrogen values, there is sufficient evidence which suggests that aromatase inhibitors can improve and increase recovery rates.

Unsuccessful PCT:

In some cases the aforementioned post cycle therapy protocols as well as those which are not mentioned may be unsuccessful in the restoration of homeostasis. This should not warrant immediate concern. Many endocrinologists have concluded that the only form of treatment in this particular scenario is hormone replacement therapy (HRT).

This is far from the truth. The reason many endocrinologists have come to this conclusion is due to the fact that very few of them have the experience treating severe forms of secondary acquired hypogonadotropic hypogonadism. They are unfamiliar with proper protocols which include high dosage HCG administration and additional gonadotropin preparations such as HMG or rFSH. This complication puts the patient at risk for potential and unknown side effects in the eyes of the doctor. Therefore, HRT is a reasonable solution since it will quickly alleviate the majority of the uncomfortable symptoms that the patient is experiencing.

Aside from disappointing blood work results which illustrate the typical signs of an unsuccessful recovery, the key physical indicator that the treatment is unsuccessful is testicular atrophy. In this case, HCG is continued with the necessary adjustments in dosage and frequency until an increase in testicular volume has been achieved. There is no one size fits all protocol since every case varies and deserves an individualized approach. Subsequent changes will be based upon the individual’s response to each particular stage. All the variable factors involved during the recovery process need to be considered. It’s far from accurate to reach the conclusion that HRT is needed if one specific recovery protocol is not successful.

Ongoing Argument(s):

Hypothetically speaking, if testicular function and volume have been maintained during cycle with HCG, SERMs are then utilized to counteract the imbalance in the androgen:estrogen ratio encountered post cycle as the exogenous androgens diminish. This results in the prevention of estrogenic side effects while increasing pituitary LH secretion which in turn increases testosterone production.

There is nothing wrong with using a commonly referred to protocol which recommends 250-500 IUs HCG 1-2x/wk to be incorporated throughout the cycle. However, a significant cause for concern in regards to this protocol relates to the cessation of HCG once the cycle has completed and from that point on, the only substances used during PCT are SERMs which consist of Nolvadex and/or Clomid. Realistically, there is absolutely no guarantee that this formula prevents testicular atrophy to the extent where the overall volume and function of the testes are in an optimal state. Unfortunately, a large majority of individuals do not realize or are not aware that Leydig cell desensitization does in fact occur with prolonged or high dosage HCG usage. Therefore, users which follow this protocol whom do not incorporate Nolvadex or an aromatase inhibitor are now susceptible to Leydig cell desensitization which may render HCG usage post cycle ineffective when and if needed.

During conservative cycles, there is substantial evidence which exists that supports the effectiveness of the HCG during cycle and SERMs only post cycle protocol, especially when proper estrogen and prolactin management has been incorporated. However, this conclusion is much more difficult to achieve on heavy or prolonged cycles. Testicular volume should be maintained to an acceptable extent but that does not necessarily result in an improved recovery as severe HTPA suppression still exists which is not immediately repairable through the usage of SERMs.

The most common argument here when incorporating HCG during PCT is that HCG itself is suppressive. This is true and one particular way this occurs is though the constant binding of HCG which disrupts the endogenous pulsatile secretion of LH. A recent study which included the usage of 250 mcgs Ovidrel (rHCG) 2x/wk for 12 weeks demonstrated that the patients resumed normal HPTA function within four weeks upon cessation, without the usage of SERMs. What’s even more interesting is that 250 mcgs rHCG is the equivalent of approximately 5,000 IUs uHCG. Therefore, putting things into perspective, a few additional weeks of suppression is nothing to be overly concerned about compared to and considering the 12 weeks of suppression incurred during the average cycle. The usage of HCG during PCT is a minimally intrusive variable where the benefits clearly exceed the associated costs.

Conclusion:

PCT should begin after the last injection and/or anabolic steroids intake. More specifically, a relative guideline to begin PCT is within 5-10 days when using long acting esters or 1-3 days when using short acting esters. This PCT protocol should consist of 1,000-1,500 IUs HCG 3x/wk (mod/wed/fri) in combination with 20 mgs Nolvadex and, if necessary, 50-100 mgs Clomid. The mid/intermittent cycle protocol of 500-1,000 IUs HCG and 20 mgs Nolvadex for 7 days consecutively can and should be utilized when necessary during prolonged (12+/wks) or heavy dosage (1,000+mgs/wk) cycles. In addition, blood work should be performed before beginning a cycle and after completing a cycle in order to establish baseline values and evaluate recovery, respectively.

If recovery is unsuccessful, HCG is continued with an adjustment in dosage and frequency as necessary until the increase in testicular volume and function have been achieved which is unlike the more typical, yet incorrect belief that HCG is only to be used for a short period of time. Once there is a plateau in the response to HCG, treatment with an FSH preparation such as human menopausal gonadotropin (HMG) or recombinant follicle stimulating hormone (rFSH) should be added at a starting dose of 75-150 IUs on alternate days. This continual usage is not necessary and avoidable in most cases by utilizing the mid/intermittent protocol previously mentioned, but it is much more common and less avoidable with long term (1+/yr) users, whom have not taken the suggested preventive measures, and/or improper recovery from previous cycles regardless of which protocol is chosen.

With the usage of HCG post cycle, your androgens are elevated but well below that of supraphysiological concentrations from exogenous hormones. In addition, a noteworthy difference is that the effect is through a direct stimulation of testicular production compared to the secondary nature of SERMs in conjunction in the presence of testis that are not guaranteed to be in an optimal functioning state. Upon completion, blood work will display significantly higher levels of LH, FSH and testosterone in this environment which includes HCG and SERMs during PCT versus HCG during cycle and SERMs only during PCT. This ultimately results in a more comfortable as well as tolerable recovery both physically and psychologically. In conclusion, HCG should always be included during PCT in combination with SERMs regardless of what protocol has been utilized during cycle to prevent testicular atrophy, in order to achieve an optimal recovery.

Albuterol VS Clenbuterol

Clenbuterol has been used for years for its ability to shed body fat and preserve lean muscle mass. Although this is highly debated, Clenbuterol has been thought to have mild anabolic properties, which is believed to be the reason for the preservation of muscle mass when dieting. Bodybuilders have used Clenbuterol as a diet drug for over three decades, and until the last 10 years or so.

Since Albuterol has become a popular drug with many bodybuilders trying to lose body fat, there have been many different views when comparing Clenbuterol and Albuterol. Both drugs are beta-2 adrenoreceptor agonists, which act on the beta-2 adrenergic receptor, causing a dilation of bronchial passages, vasodialation in muscle and liver, and a release of insulin. These characteristics have made this class of drugs a prime choice in treating asthma.

When comparing these two drugs, we must understand how they work, which requires taking a look at the beta-adrenergic system. This system is comprised of adrenoreceptors, but what is relevant here are the beta-receptors. The beta-receptors are located in the cell’s phospholipid membrane, and are stimulated by stimulants like clenbuterol, albuterol, and ephedrine. The beta-receptors can be divided into three subtypes, but when looking at clenbuterol and albuterol, the beta-2 receptor is what we are focusing on. Stimulation of the receptors from stimulants like Clenbuterol or Albuterol causes a breakdown of fatty acids into the bloodstream to be used as fuel. With this stimulation there will be an increase in heart rate and body temperature.

Beta-2 adrenoreceptor agonists, such as Clenbuterol and Albuterol, have been shown to activate cyclic-Adenosine Monophosphate (cAMP). cAMP then activates calpistatin, which inhibits calpain. Calpain degrades protein in muscle tissue. By inhibiting calpain, beta-2 adrenoreceptor agonists have been shown to be anti-catabolic. This becomes very beneficial when trying to shed body fat while wanting to preserve muscle mass. A bodybuilder’s goal is always to become as lean as possible while holding as much muscle mass as possible. This is why this class of drugs has always been very popular among this group.

Albuterol can be viewed as a shorter acting version of Clenbuterol. They both stimulate the beta-receptors in a similar manner. When looking at beta-2 agonists that are longer acting than Clenbuterol, and then compared to Clenbuterol, Clen had a better rating when it came to anabolic effects. So it would be logical to think that maybe Albuterol has an even higher anabolic effect than clenbuterol due to the shorter half-life, but it isn’t always that simple. There is still much more that needs to be studied and looked at before determining this.

The anti-catabolic effects of Clenbuterol and Albuterol are a plus, but the primary goal of taking either drug is for fat loss. When honestly looking at things, there are far too many other substances that carry more weight in preserving muscle mass than any beta-2 agonist when there is a deficit in calories. Anabolic steroids and human growth hormone cast a shadow over Clenbuterol or Albuterol when it comes to preserving lean muscle mass. Most bodybuilders that are dieting are usually taking one or both of these substances, so the anti-catabolic effect is not much of a factor compared to the fat loss that occurs from either of these drugs.

When comparing Clenbuterol and Albuterol in the category of fat loss, the half-life has to be looked at. Clenbuterol has a half-life of about 48 hours, while Albuterol has a half-life of just about 6 hours. There is an upside and downside to the half-life to both of these drugs. The long half-life associated with Clenbuterol will allow a person to dose less often with the drug still staying active in the system and doing its job with burning body-fat. The downside to this long half-life is that the body stops responding to this drug fairly quickly. This requires a person to cycle the drug 2 weeks on and 2 weeks off, or the person will have to dramatically increase the dose after 2 weeks to feel any effect at all. The short half-life of Albuterol requires a person to have to dose more often throughout the day for the drug to be most effective, but tolerance is not built up nearly as fast as it is with Clenbuterol.

Albuterol has a more mild stimulant effect than Cenbuterol, and some people find it easier to focus while taking albuterol than Clenbuterol. Clen leaves some people shaky, sweaty, and unable to focus during a workout, so albuterol would be the obvious choice. When dieting, some people feel sluggish and find it hard to power though a workout without a strong stimulant. Clenbuterol will often give them that extra edge to push through a workout they normally couldn’t while dieting. Clenbuterol would be the choice for this group.

Both of these drugs fall into the same class of drugs. There are positives and negatives to each one, but when it comes to Clenbuterol and Albuterol, everything kind of evens out at the end of the day. Everyone responds differently to every compound. Some are more sensitive to stimulants than others. I do believe that you will not see much difference in the fat burning and anti-catabolic effects between Clenbuterol and Albuterol.

Always Add Proviron Into Your Cycle

Proviron is an interesting and often overlooked bodybuilding drug. It’s an orally active form of DHT (Dihydrotestosterone) and as such, a Proviron cycle delivers all the good and all the bad that DHT has to offer the bodybuilder.

As for the good, DHT from a Proviron cycle is a pure androgen, but unlike testosterone, DHT doesn’t cause any water retention. DHT is what makes muscle “hard.” And it lowers SHBG (Sex hormone Binding Globulin) which prevents estrogen from forming. By preventing estrogen from forming, DHT can be used to prevent the aromatization of other steroids. This means that DHT from a Proviron cycle can keep steroids in the testosterone family from converting to estrogen and causing in men estrogen related side effects like gynecomastia. DHT also contributes to erectile rigidity. All good stuff! There are drawbacks however, most notably prostate hypertrophy and hair loss. So one may say DHT is pure maleness.

In the past, DHT was regarded as the “bad” form of testosterone and there have been many products available to reduce it. 

So, we know DHT can be tricky and we know we need it. But, where does Proviron fall into the equation? Well, proviron can be a nice addition to any cycle for a variety of reasons. Since it lowers SHBG it allows for more total testosterone to become bioavailable. Up to 90% of testosterone, be it natural or administered, remains in a “bound” state (due to SHBG) rendering it useless for building muscle. Proviron releases more active testosterone into the bloodstream so you can get more of its benefits.

Proviron also works well as an anti estrogen, in fact, it’s superior to most traditional anti e’s such as Nolvadex because there is no rebound effect. Instead of removing estrogen, proviron prevents the formation of estrogen in the first place.

Proviron is excellent for contest preparation because it adds density and definition. You see, androgen receptors are found in your fat cells as well as muscle cells and DHT binds so well that there’s a distinct fat burning effect from its use.

Many people use Proviron in between cycles to maintain muscle gains and libido. However, DHT will not do much in regard to maintaining muscle. And as with any drug, a tolerance is developed over time so using Proviron to help a lagging libido will just add to further suppression once the use of the drug has ceased.

The main downside of Proviron is that since it contains no anabolic properties, it will not result in much muscle growth if used alone. It truly is a “kicker” to other steroids, most notably testosterone. And although it doesn’t cause water retention, it can increase blood pressure. DHT can also increase irritability if duration exceeds more than a few weeks.

There have recently been advances in natural alternatives to Proviron. Avenacosides have been shown to lower SHBG and to raise free testosterone by as much as 20%. Since Proviron is used mostly as an ancillary substance and does not have much of an effect of actual growth, more and more people are opting to go the natural route. Although Avenacosides won’t work quite as well, they are effective and can be used in conjunction with a lower dose of Proviron for an even more powerful muscle hardening/libido enhancing effect.

What is Clenbuterol?

Clenbuterol is a bronchodilator used to threat asthma and other conditions. It belongs to the sympathomimetics group of drugs, which affect sympathetic nervous system, mostly by affecting beta or alpha receptors. In Clenbuterol’s specific case, its effect results on increased fat loss.

How does it works?

Being a beta-2 agonist, Clenbuterol works as a fat burner by binding with beta receptors (mostly type 2 in humans) in fat and muscle tissue in the body. When this bond is formed, the receptors then start a series of chemical reactions leading to the production of cAMP. cAMP then produces and activates enzymes which induce fat breakdown.
Given that Clenbuterol has little effect on beta-1 receptors, it is capable of reducing reversible airway obstruction, with less cardiovascular effects, if compared with nonselective agonists. This whole process will create an increase in the body’s temperature, which will make the body burn more calories. However, your body will attack this matter through beta receptor down regulation, which will dictate how long can you run clenbuterol and still get results. Clenbuterol also lowers lipoprotein lipase activity. This prevents fat deposition and makes fat more responsive to the Hormone Sensitive Lipase activity described above. It is also important to mention, that beta-2 agonists such as Clenbuterol have been shown to increase T3 levels, thus enhacing clenbuterol’s fat burning capabilities.

Another benefit of Clenbuterol, are its anti-catabolic properties. This occurs as Clenbuterol blocks both ca++ dependent proteolysis in rat skeletal muscle, as well as the ubiquitin-proteasome proteolytic pathway. When beta 2 blockers are administered, the effect is not observed. Blocking these pathways inhibits muscle tissue breakdown.

One important thing that must be observed, is that in the past it was believed that Clenbuterol could be used as an Anabolic in humans. This was based on several studies performed on livestock and rats, which in fact showed such results. However, the doses used on these animals, if translated into human doses, would probably kill a person.

How is Clenbuterol dosed

For males the common maintenance dose of Clenbuterol is between 120-140mcgs per day. For females the dose of Clenbuterol goes down to around 80-100mcgs per day. It should however taken seriously, and each person must stablish a maximum safe dose, based on their experience.

How should I cycle Clenbuterol?

This is a very important point of every drug, that some pleople overlook, or just don’t go into enough detail. Clenbuterol should be cycled 2 weeks on/2 weeks off (this can change if ketotifen is used-see below). This is caused by two major factors.

  1. Beta-2 receptor down regulation: this will cause the effectiveness of Clenbuterol to be reduced as time goes by.
  2. Long half life: it has biphasic elimination, with the half-life of the rapid phase being about 10 hours, and the slower phase being several days. This cause the theory of using it 2 days on/2 days off to flaw, as in fact you would be on the whole time, and your beta receptors would not have enough time to recover.

If you use ketotifen you could in theory stay on Clenbuterol for an undefined period of time. This is because contrary to Clenbuterol, ketotifen up regulates beta receptors. The recommended dose of ketotifen is 2-3mg per day, and it should be taken before bed, as it causes drowsiness.

A typical Clenbuterol cycle for a male would be something like this:

  • Day 1: 20mcgs
  • Day 2: 40mcgs
  • Day 3: 60mcgs
  • Day 4: 80mcgs
  • Day 5: 100mcgs
  • Days 6-14: 120mcgs

Please be aware that you must set your own limits, and listen to your body.

What are the possible side effects?

Common side effects of Clenbuterol are increased perspiration, insomnia, muscle spams, restlessness, palpitations, involuntary trenbling of the fingers, nausea and increased blood pressure

Dealing with Injection Pain

Injection is not the most popular method of administration for those new to steroids, but as they will quickly learn, injectables are the safest for men in terms of long term health and by following proper injection instructions, the risks are minimal. However, with this in mind, injections can be difficult as it may result in pain and even infections. In this article we will examine some of the reasons this happens and ways to avoid it.

Causes of Injection Pain
One of the main reasons injections can be painful is the esters involved. Esters are basically buffers that slow the release of a steroid into your system and this allows for less vigorous injection cycles. The pain is caused after the steroid has been released leaving just the ester to crystallize which makes it take longer to dissolve while longer esters dissolve more readily. This is just one reason why it is a good idea to alternate your injection site as you do not want these crystals to build up and cause pain. Additionally, steroids that are released quickly can also cause pain, the worst offender being water based injections or injections with no ester at all. Additionally, injecting too quickly can tear muscle tissue while injecting into a new spot can be painful as the muscle will not absorb the compound as quickly.

Methods of Relief
One method of alleviating pain is to add additional sterile filtered oil to your injections at a ratio of 50:50 and injecting slowly (30 seconds per ml). One may also take some ibuprofen to decrease any swelling. Another strategy is to freeze the tip of the needle thus decreasing the pain as it pierces the skin.

In addition to all of these strategies, it is also important to keep everything you are using clean, to choose the right needles, to pick the right injection sites, and follow the proper injection guidelines accordingly in order to avoid infection. More information on these topics can be found here.

Infection
Infections can be identified by these localized four traits: heat, pain, redness, and swelling. You may also experience chills, fever, and pus excretion. If you believe you may have an infection, you should see your doctor or local hospital immediately. Although it can be a good idea to keep a various array of antibiotics at home, unless you see fast results you may risk giving the infection enough time to enter your bloodstream. Also, the misuse of antibiotics can create immunity in the bacteria making it resistant to treatment and therefore a threat to others. Always talk to a doctor when administering an injection, it could save your life.

Risks/Side Effects of Trenbolone

Trenbolone does not exhibit any estrogenic activity and therefore estrogenic side effects are not a concern with this compound. It is also resistant to the 5 alpha reductase enzyme, but this is of little comfort to a user as trenbolone is already of the most androgenic drugs in common use by steroid users. For this reason androgenic side effects should be expected by most users that undertake a cycle of this drug. Prostate enlargement and oily skin/acne are commonly reported by users. As well anecdotally many users have reported that trenbolone is one of, if not the, harshest compound for losing one’s hair. If a user is genetically predisposed to male pattern baldness he may want to avoid trenbolone.

Having listed the harsh androgenic nature and side effects associated with trenbolone, it should come as no surprise that women are not recommended to use this compound. The usual virilizing effects such as deepening of the voice, body/facial hair growth, and enlargement of the clitoris, among others are likely to cause problems for female users. These effects can appear at even relatively low doses. Trenbolone is not a compound that women should attempt to administer.

Now due to the lack of estrogenic side effects associated with trenbolone it would seem that users would have little to worry about in terms of side effects like gynecomastia, water retention, etc. However trenbolone is a progestin, meaning that it has the ability to bind to receptors of the female sex hormone progesterone. Also, like other 19-nor compounds trenbolone increases prolactin levels. Side effects related to these reactions can include breast growth and lactation. To prevent these side effects as they relate to increased prolactin levels a user can use several compounds including bromocriptine, vitamin b6, and/or cabergoline. Letrozole can also be used to lower progesterone levels. It should also be noted that trenbolone lowers thyroid levels temporarily which in turn raises prolactin levels. It is therefore advisable that users may want to use the compound T3 to combat this effect in part.

Being a progestin, trenbolone also has a dramatic effect on users’ natural testosterone production. Much in the same way that nandrolone does, trenbolone can suppress the natural production of testosterone for weeks after a user has ceased administering it. For this reason it is advisable that users use testosterone in conjunction with trenbolone if they wish to avoid sexual dysfunction, libido problems, or mental side effects associated with a lack of testosterone. Anecdotally many users have also reported that testicular atrophy is nearly always a problem when using trenbolone and that it is much more dramatic than with other compounds. Users may wish to administer human chorionic gonadotropin to help counteract this.

The psychological effects of trenbolone use are also quite distinct in some users. Of course the obvious effect would be a reaction to the androgenic nature of the compound. An increase in aggressiveness is often reported by users, as androgens help to affect brain chemistry and may cause feelings of well-being, angst, aggression, or anxiety. As well, anecdotally some users have also reported that they experience vivid dreams while using the compound.

Kidney and liver function is negatively affected by trenbolone use as well. It is recommended that users closely monitor these throughout a cycle making sure that no problems arise. As well, users will also report that darker than normal urine is a common side effect of use of trenbolone. This should not be a cause of alarm among users; however they should ensure that there is no blood in the urine as this is obviously a sign of trouble.

Comparison between Trenbolone Enanthate and Trenbolone Acetate

Trenbolone enanthate unlike testosterone-based steroids, does not result in the development of feminine sexual characteristics in men. This steroid has the ability of stimulating protein synthesis gains that prove useful in new tissue formation that, in turn, promote muscle growth or hypertrophy. Moreover, Trenbolone enanthate is used by those into boxing, MMA, and cycling as it helps with strength and speed. Trenbolone enanthate can demonstrate unmatched efficacy in reducing body fat levels as it has a strong cortisol-reducing effect along with the ability to bind to the glucocorticoid receptor.

Trenbolone Acetate

Trenbolone Acetate has the molecular formula of C20H24O3 and has an anabolic-androgenic ratio of 500:500. It has an active life of 2-3 days and has the molecular weight of 270.3706 g/mol at the base. Trenbolone Acetate, a 19-nor steroid, is derived from the compound Nandrolone. It is characterized by strong androgenic properties and no estrogenic activity. Trenbolone Acetate has a binding affinity for androgen receptor five times the strength Testosterone. Trenbolone acetate is admired globally by elite athletes as it can dramatically improve the uptake of nitrogen by muscles and improve the level of protein synthesis. Ideal for cutting cycles, Trenbolone Acetate does not get metabolized by aromatase or 5α-reductase into estrogenic compounds like estradiol or into Dihydrotestosterone (DHT).

The primary difference between Trenbolone Acetate and Trenbolone enanthate is esters. While Trenbolone enanthate, featured by comparatively less esters, peaks at a slow pace and leaves the system slowly, Trenbolone Acetate with more esters peaks faster and leaves the system faster. Moreover, Trenbolone Acetate is better when it comes to maintaining stable blood levels, especially when athletes want to gain muscle mass and strength when following a dieting regimen. Furthermore, the human body finds it simpler to absorb a higher percentage of milligrams when injected with the acetate form rather than in the enanthate form.

A big majority of athletes and bodybuilders, especially those into elite sports and professional bodybuilding, prefer Trenbolone acetate over Trenbolone enanthate. This is because the enanthate version of Trenbolone doesn’t result in as much hardening of muscles as experienced with the acetate version. While the acetate version is known to promote cutting, the enanthate version is used for adding muscle size. Beginners to the world of anabolic compounds should opt for Trenbolone Acetate instead of Tren E as Tren acetate gets out of the system quickly. However, the enanthate version of Tren is rarely associated with night sweats, insomnia , and over-aggression and allows athletes to reap the optimum benefits of the potent steroids. Trenbolone enanthate is less likely to result in the dreaded Tren cough and involves less pinning but the use of this steroid involves more of patience as results can take time.

The fact that Trenbolone acetate is easily and more readily available puts it ahead of the enanthate version. In addition, this short ester compound is better than enanthate version when it comes to controlling and manipulating blood levels.

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