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Trenbolone (Acetate / Enanthate). Tren Steroid Cycles and Dosages

Trenbolone is a very powerful steroid that was originally manufactured as pellets that were administrated beneath cattle skin, later to be brewed by underground labs for use by anabolic steroid users. Trenbolone has never been approved by the FDA for use in humans, and the drug is commercially used as vetinary finaplex pellets. Trenbolone is now widely available via many underground labs, and is now a very popular steroid due to its very strong and potent effects.

A highly anabolic and androgenic steroid, Trenbolone can be a very harsh steroid for users that are prone to androgenic related side effects, and even those who have previously not been. Trenbolone has been said to bind to the androgen receptor at a rate as highly as three times that of testosterone, so we can strongly expect androgenic related side effects to result from its use in users that are prone to such side effects. Acne can be expected in prone users, and those who often have a break out of acne from use of other steroids, such as testosterone products, may wish to think about running a different compound. Accutane is sometimes taken with Trenbolone to combat the acne. Trenbolone will almost certainly speed up male pattern baldness (MPB) due to its very strong androgenic effects, and therefore will not be a suitable steroid for those who are prone to MPB and wish to avoid it.

Sweating and a decrease in cardiovascular capability is often widely reported with Trenbolone usage. Trenbolone is not really suitable for those who participate in sporting activities that require cardiovascular fitness. Sweating is also reported greatly at night time, with insomnia and strange dreams. Water intake should be efficient when taking Trenbolone due to water loss.

Although Trenbolone does not aromatise, in the present of oestrogen Trenbolone can manifest progesterone related side effects, such as gynecomastia. This should be kept in mind when stacking with other androgenic anabolic steroids, such as testosterone. Trenbolone is also very harsh on the HTPA function, and recovery is often said to be harsher compared to other steroids. The use of HCG is often advised for this reason.

Trenbolone (Tren) cough is a widely experience side effect from Trenbolone usage, but the cough has also been reported in the use of other anabolic steroids. The cough itself is not a pleasant experience, usually starting with a strange taste in the mouth followed by a strong and uncontrollable cough. What causes Tren cough is a much disputed topic in the bodybuilding community, with claims of it being caused the amount of Benzyl Alcohol used in its production by the labs, or by the oxidised partials that get into the blood stream of the user upon administration of the Trenbolone.

Users can expect rapid gains in muscle size and strength from the use of Trenbolone. The steroid is often use in cutting cycles due to its ability to cause hardening of the muscles, and positively changing a persons body composition. Trenbolone is also said to be responsible for some level of fat loss, but we should not rely on the compound for this benefit. Novice users may wish to use the faster Acetate ester due to its faster half life, and therefore the ability to cause a quicker drop in blood levels upon ceasing the drug incase any undesirable side effects become present. 75mg-100mg each other day (EOD) is common, and is often stacked with testosterone propionate. Greater dosages is sometimes administrated by more advanced users, often 500mg + per week.

Due to its high androgenic characteristics, Trenbolone is not really a suitable steroid to be taken by women.

Trenbolone is a very powerful and potent androgenic anabolic steroid produced by many respected underground labs. For those who can handle the harsh side effects it can be a very effective drug, possessing the ability to drastically change a users body composition.

Trenbolone anabolic steroid cycles

Novice user’s cycle

  • 75-100mg of Trenbolone each other day (EOD) for 8 weeks, stacked with Testosterone Propionate 100mg each other day (EOD). Start pct 4 days after last injection

Intermediate/Advanced user’s cycle

  • 500mg of Trenbolone stacked with 500mg-1000mg of Testosterone Enanthate per week for 10+ weeks.

Trenbolone is often used in competitors cuttings stacks prior to content, and stacked with many other anabolic steroids.

Post Cycle Therapy

Pulsatile secretion of gonadotropin releasing hormone (GnRH) from the hypothalamus is required for both the initiation and maintenance of the reproductive axis in the human. Pulsatile GnRH stimulates the biosynthesis of luteinizing hormone (LH) and follicle stimulating hormone (FSH) that in turn initiates endogenous testosterone production and spermatogenesis as well as systemic testosterone secretion and virilization. Failure of this episodic GnRH secretion or disruption of gonadotropin secretion results in the clinical syndrome of hypogonadotropic hypogonadism (HH).

The usage of anabolic steroids may result in a functional form of HH known as Secondary Acquired Hypogonadotropic Hypogonadism and is diagnosed in the setting of a low testosterone level and sperm count in association with low or inappropriately normal serum LH and FSH concentrations.

In order to avoid any unnecessary confusion, it is important to understand what the actions of Gonadatropin therapy and Selective Estrogen Receptor Modulators are as well as how they differ from each other and more specifically, during post cycle recovery (PCT).

Gonadotropin Therapy:

There is nothing more effective than Human Chorionic Gonadotropin (HCG). The action of HCG is identical to that of pituitary LH. This takes place independently and is not affected by exogenous hormones and/or preexisting HPTA suppression. Therefore, it directly stimulates a dramatic increase in endogenous testosterone production, spermatogenesis and testicular volume. The primary goal during the first few weeks of PCT is to quickly restore testicular volume and function. Also, the dramatic increase in testosterone production is necessary to avoid and/or minimize the unfavorable “crash” effect. In the majority of individuals with larger testes at baseline, HCG alone is sufficient in restoring endogenous testosterone production as well at the induction of spermatogenesis which is most likely a result of residual FSH secretion. Once there is a plateau in the response to HCG, treatment with an FSH preparation such as human menopausal gonadotropin (HMG) or recombinant follicle stimulating hormone (rFSH) should be added in combination to HCG.

*The addition of an FSH preparation is rarely required and is best suited for severe cases of HH. FSH preparations are not readily available to most individuals. Therefore, there is no need to go into details with respect to its application at this time.

HCG is administered by subcutaneous (SC) or intramuscular (IM) injection. The average (3ml 22-25g x ?-1½”) syringe is adequate for IM injections but insulin syringes (½-1ml 28-30g x ½-1”) are recommended for SC injections. In regards to effectiveness, there should be no discernable difference between either of the techniques. The individual should opt for the most comfortable and/or convenient form of administration.

HCG ampoules are supplied in 500, 1,000, 2,000, 5,000 and 10,000 IU preparations accompanied by 1 ml of sterile dilluent. It should be stored at a controlled room temperature (15-30 degrees C or 59-86 degrees F) and should be used immediately after reconstitution.

HCG multidose vials are supplied in 2,000, 5,000 and 10,000 IU preparations accompanied by 10 ml of bacteriostatic water. It should be stored at a controlled room temperature (15-30 degrees C or 59-86 degrees F), refrigerated (2-8 degrees C or 36-46 degrees F) after reconstitution and used within 30 days.

The terms international units (IUs) can occasionally cause confusion when reconstituting and measuring HCG. The actual process is quite elementary and the concentration per ml (cc) is dependant on the concentration of the lyophilized powder and the volume of dilluent used for reconstitution. For example, if you dilute 5,000 IUs HCG with 5ml (cc) solvent, the end result is 1,000 IUs per ml (cc). Divide the same 5,000 IUs with 10 ml (cc) and the end result is 500 IUs per ml (cc).

*Bacteriostatic water should always be utilized during reconstitution when long term (30 day) storage and multi dose administration are required.

Selective Estrogen Receptor Modulators:

Selective estrogen receptor modulators (SERMs) such as Clomiphine (Clomid) and Tamoxifen (Nolvadex) increase pituitary LH secretion in secondary manner by blocking estrogen negative feedback on the HPTA. On average, this is not strong enough by itself to counteract the severe imbalance of the androgen:estrogen ratio that is encountered post cycle, especially in the presence of testicular atrophy. Therefore, SERMs are used during PCT primarily as an anti estrogen and to continue the stimulation of pituitary LH after HCG has been discontinued.

Nolvadex is widely available in 10 mg or 20 mg tablet preparations and Clomid is available in 50 mg tablet preparations.

Before Beginning PCT:

It is highly recommended to establish baseline blood values before beginning a cycle. The same principle applies to establishing post cycle blood values, which are necessary for evaluating recovery. Post cycle blood work should be obtained approximately 4 weeks after the cessation of PCT in order to determine accurate readings. Additional blood work should be performed when applicable and/or required.

The following are Fasting blood values:

Hormone

  1. Cortisol, Total
  2. Estradiol, Extraction
  3. Prolactin
  4. LH
  5. FSH
  6. T3, Free
  7. T4, Free
  8. TSH
  9. Testosterone, Total, Free and Weakly Bound
  10. Hemoglobin A1C
  11. Fasting Insulin
  12. Somatomedian C (optional)

Cardiovascular

   13. CBC
   14. Comprehensive Metabolic Panel
   15. Lipid Panel

Other

    16. GGT Important Liver Value not included in Comp Metabolic Panel

When to begin PCT:

On average, begin PCT approximately 5-10 days after your last injection regardless of longer acting esters. Begin PCT 1-3 days after your last injection and/or intake when using short acting esters.

Keep in mind, pituitary LH secretion automatically increases as the hormones diminish from your system. The elevated androgen levels are from an exogenous source and your endogenous production is suppressed. Therefore, waiting for the exogenous androgens to completely clear from your system, ultimately results in lower total concentrations of androgens in your system when beginning PCT. This leads to an unfavorable andgrogen:estrogen ratio and the well known “crash” effect.

*As previously mentioned, the actions of HCG take place independently and is not affected by exogenous hormones and/or preexisting HPTA suppression. There are no contradictions with respect to the effectiveness of HCG usage while exogenous hormones are present in your system.

PCT Protocol(s):

1.) 1,000 IUs HCG 3x/wk (mon/wed/fri) in combination with 20 mgs Nolvadex for the first 3 weeks. After, discontinue HCG and continue with 20 mgs Nolvadex for an additional 3 weeks.

2.) 1,000 IUs HCG 3x/wk (mon/wed/fri) in combination with 20 mgs Nolvadex and 50 mgs Clomid for the first 3 weeks. After, discontinue HCG and continue with 20 mgs Nolvadex and 50 mgs Clomid for an additional 3 weeks.

3.) 1,500 IUs HCG 3x/wk (mon/wed/fri) in combination with 20 mgs Nolvadex for the first 3 weeks. After, discontinue HCG and continue 20 mgs Nolvadex for an additional 3 weeks.

4.) 1,500 IUs HCG 3x/wk (mon/wed/fri) in combination with 100 mgs Clomid and 20 mgs Nolvadex for the first 3 weeks. After, discontinue HCG and continue with 50 mgs Clomid and 20 mgs Nolvadex for an additional 3 weeks.

Option one can be considered as a standard PCT protocol. This should apply to all basic cycles. Option 2 is generally the same as option one except for the addition of Clomid which is added as a supporting recovery aid. Option three and four incorporate a higher HCG dosage and have a relationship similar to options one and two in the sense that Clomid is incorporated in the latter as a supporting recovery aid.

*The majority of my experience is with intermediate to advanced athletes whom have completed multiple cycles with higher dosages. Therefore, based upon previous blood work results and considering the common or convenient preparations available, we have established that 1,500 IUs 3x/wk (mon/wed/fri) to be the optimal HCG dosage to begin with. The Nolvadex dosage remains unchanged however Clomid is utilized throughout the entire PCT at 100 mgs during the first 3 weeks and 50 mgs for the last 3 weeks.

HCG During Cycle:

HCG in combination with Nolvadex can and should be used during prolonged (12+/wks) and high dosage (1,000+mgs/wk) cycles. In this case, 500-1,000 IUs HCG in combination with 20 mgs Nolvadex for 7-10 days consecutively is administered mid cycle or intermittently (every 6-8 weeks) during the cycle.

Maintaining testicular volume during cycle does in fact improve recovery when compared to atrophied testes when beginning PCT. This solution addresses both testicular atrophy and prevention of Leydig cell desensitization (discussed next) associated with HCG usage.

Leydig Cell Desensitization:

Leydig cell desensitization does in fact occur to some degree with prolonged or high dose HCG usage. Using it continuously during a cycle could possibly cause the LH receptor to desensitize which in turn would ultimately render the PCT to be either less effective or possibly useless. This seems counterproductive. HCG will not be needed on cycles where the proper ancillaries are used and where the dosages/durations are realistic.

The previous summary was a general statement. The reality and good news is that Leydig cell desensitization due to HCG usage is blocked and/or minimized by Nolvadex. This occurs by suppressing HCG’s ability to inhibit the conversion of 17 alpha hydroxyprogesterone to testosterone.

Additional Factors That Influence Recovery:

Factors that may complicate and/or delay recovery are elevated levels of estrogen and prolactin. Both of these hormones, when elevated, exert negative feedback on the HPTA. Estrogen and its side effects can be controlled by using an aromatase inhibitor such as Aromasin, Femara and Arimidex during cycles including aromatizing anabolic steroids. Prolactin and its side effects can be controlled by using an anti Prolactin during cycles containing nandrolones. If these measures have not been addressed during the cycle, they will more than likely need to be addressed during PCT. In this scenario, the objective is to lower these hormones to acceptable levels in order to avoid the complications and/or delay in recovery. Blood work is imperative in evaluating the effectiveness of therapy. This will provide a clear and concise answer in regards to the adjustment of dosages and continuation of medication if necessary.

*There are numerous studies which support and refute the association of nandrolones and prolactin. However, based on first hand experience and blood work results, there are far more individuals today whom can testify that the usage of nandrolones can attribute to an increase in prolactin concentrations. In addition, many individuals have reported elevated prolactin levels during cycles which do not contain nandrolones. The common factor within these cases is supraphysiological levels of estrogen. Estrogens act directly at the pituitary level by causing the stimulation of lactotrophs which in turn enhances prolactin secretion. This is another reason why estrogen management in the form of an aromatase inhibitor should be included with cycles containing aromatizing anabolic steroids. Although not absolutely necessary and considering the necessary restoration of physiological estrogen values, there is sufficient evidence which suggests that aromatase inhibitors can improve and increase recovery rates.

Unsuccessful PCT:

In some cases the aforementioned post cycle therapy protocols as well as those which are not mentioned may be unsuccessful in the restoration of homeostasis. This should not warrant immediate concern. Many endocrinologists have concluded that the only form of treatment in this particular scenario is hormone replacement therapy (HRT).

This is far from the truth. The reason many endocrinologists have come to this conclusion is due to the fact that very few of them have the experience treating severe forms of secondary acquired hypogonadotropic hypogonadism. They are unfamiliar with proper protocols which include high dosage HCG administration and additional gonadotropin preparations such as HMG or rFSH. This complication puts the patient at risk for potential and unknown side effects in the eyes of the doctor. Therefore, HRT is a reasonable solution since it will quickly alleviate the majority of the uncomfortable symptoms that the patient is experiencing.

Aside from disappointing blood work results which illustrate the typical signs of an unsuccessful recovery, the key physical indicator that the treatment is unsuccessful is testicular atrophy. In this case, HCG is continued with the necessary adjustments in dosage and frequency until an increase in testicular volume has been achieved. There is no one size fits all protocol since every case varies and deserves an individualized approach. Subsequent changes will be based upon the individual’s response to each particular stage. All the variable factors involved during the recovery process need to be considered. It’s far from accurate to reach the conclusion that HRT is needed if one specific recovery protocol is not successful.

Ongoing Argument(s):

Hypothetically speaking, if testicular function and volume have been maintained during cycle with HCG, SERMs are then utilized to counteract the imbalance in the androgen:estrogen ratio encountered post cycle as the exogenous androgens diminish. This results in the prevention of estrogenic side effects while increasing pituitary LH secretion which in turn increases testosterone production.

There is nothing wrong with using a commonly referred to protocol which recommends 250-500 IUs HCG 1-2x/wk to be incorporated throughout the cycle. However, a significant cause for concern in regards to this protocol relates to the cessation of HCG once the cycle has completed and from that point on, the only substances used during PCT are SERMs which consist of Nolvadex and/or Clomid. Realistically, there is absolutely no guarantee that this formula prevents testicular atrophy to the extent where the overall volume and function of the testes are in an optimal state. Unfortunately, a large majority of individuals do not realize or are not aware that Leydig cell desensitization does in fact occur with prolonged or high dosage HCG usage. Therefore, users which follow this protocol whom do not incorporate Nolvadex or an aromatase inhibitor are now susceptible to Leydig cell desensitization which may render HCG usage post cycle ineffective when and if needed.

During conservative cycles, there is substantial evidence which exists that supports the effectiveness of the HCG during cycle and SERMs only post cycle protocol, especially when proper estrogen and prolactin management has been incorporated. However, this conclusion is much more difficult to achieve on heavy or prolonged cycles. Testicular volume should be maintained to an acceptable extent but that does not necessarily result in an improved recovery as severe HTPA suppression still exists which is not immediately repairable through the usage of SERMs.

The most common argument here when incorporating HCG during PCT is that HCG itself is suppressive. This is true and one particular way this occurs is though the constant binding of HCG which disrupts the endogenous pulsatile secretion of LH. A recent study which included the usage of 250 mcgs Ovidrel (rHCG) 2x/wk for 12 weeks demonstrated that the patients resumed normal HPTA function within four weeks upon cessation, without the usage of SERMs. What’s even more interesting is that 250 mcgs rHCG is the equivalent of approximately 5,000 IUs uHCG. Therefore, putting things into perspective, a few additional weeks of suppression is nothing to be overly concerned about compared to and considering the 12 weeks of suppression incurred during the average cycle. The usage of HCG during PCT is a minimally intrusive variable where the benefits clearly exceed the associated costs.

Conclusion:

PCT should begin after the last injection and/or anabolic steroids intake. More specifically, a relative guideline to begin PCT is within 5-10 days when using long acting esters or 1-3 days when using short acting esters. This PCT protocol should consist of 1,000-1,500 IUs HCG 3x/wk (mod/wed/fri) in combination with 20 mgs Nolvadex and, if necessary, 50-100 mgs Clomid. The mid/intermittent cycle protocol of 500-1,000 IUs HCG and 20 mgs Nolvadex for 7 days consecutively can and should be utilized when necessary during prolonged (12+/wks) or heavy dosage (1,000+mgs/wk) cycles. In addition, blood work should be performed before beginning a cycle and after completing a cycle in order to establish baseline values and evaluate recovery, respectively.

If recovery is unsuccessful, HCG is continued with an adjustment in dosage and frequency as necessary until the increase in testicular volume and function have been achieved which is unlike the more typical, yet incorrect belief that HCG is only to be used for a short period of time. Once there is a plateau in the response to HCG, treatment with an FSH preparation such as human menopausal gonadotropin (HMG) or recombinant follicle stimulating hormone (rFSH) should be added at a starting dose of 75-150 IUs on alternate days. This continual usage is not necessary and avoidable in most cases by utilizing the mid/intermittent protocol previously mentioned, but it is much more common and less avoidable with long term (1+/yr) users, whom have not taken the suggested preventive measures, and/or improper recovery from previous cycles regardless of which protocol is chosen.

With the usage of HCG post cycle, your androgens are elevated but well below that of supraphysiological concentrations from exogenous hormones. In addition, a noteworthy difference is that the effect is through a direct stimulation of testicular production compared to the secondary nature of SERMs in conjunction in the presence of testis that are not guaranteed to be in an optimal functioning state. Upon completion, blood work will display significantly higher levels of LH, FSH and testosterone in this environment which includes HCG and SERMs during PCT versus HCG during cycle and SERMs only during PCT. This ultimately results in a more comfortable as well as tolerable recovery both physically and psychologically. In conclusion, HCG should always be included during PCT in combination with SERMs regardless of what protocol has been utilized during cycle to prevent testicular atrophy, in order to achieve an optimal recovery.

Albuterol VS Clenbuterol

Clenbuterol has been used for years for its ability to shed body fat and preserve lean muscle mass. Although this is highly debated, Clenbuterol has been thought to have mild anabolic properties, which is believed to be the reason for the preservation of muscle mass when dieting. Bodybuilders have used Clenbuterol as a diet drug for over three decades, and until the last 10 years or so.

Since Albuterol has become a popular drug with many bodybuilders trying to lose body fat, there have been many different views when comparing Clenbuterol and Albuterol. Both drugs are beta-2 adrenoreceptor agonists, which act on the beta-2 adrenergic receptor, causing a dilation of bronchial passages, vasodialation in muscle and liver, and a release of insulin. These characteristics have made this class of drugs a prime choice in treating asthma.

When comparing these two drugs, we must understand how they work, which requires taking a look at the beta-adrenergic system. This system is comprised of adrenoreceptors, but what is relevant here are the beta-receptors. The beta-receptors are located in the cell’s phospholipid membrane, and are stimulated by stimulants like clenbuterol, albuterol, and ephedrine. The beta-receptors can be divided into three subtypes, but when looking at clenbuterol and albuterol, the beta-2 receptor is what we are focusing on. Stimulation of the receptors from stimulants like Clenbuterol or Albuterol causes a breakdown of fatty acids into the bloodstream to be used as fuel. With this stimulation there will be an increase in heart rate and body temperature.

Beta-2 adrenoreceptor agonists, such as Clenbuterol and Albuterol, have been shown to activate cyclic-Adenosine Monophosphate (cAMP). cAMP then activates calpistatin, which inhibits calpain. Calpain degrades protein in muscle tissue. By inhibiting calpain, beta-2 adrenoreceptor agonists have been shown to be anti-catabolic. This becomes very beneficial when trying to shed body fat while wanting to preserve muscle mass. A bodybuilder’s goal is always to become as lean as possible while holding as much muscle mass as possible. This is why this class of drugs has always been very popular among this group.

Albuterol can be viewed as a shorter acting version of Clenbuterol. They both stimulate the beta-receptors in a similar manner. When looking at beta-2 agonists that are longer acting than Clenbuterol, and then compared to Clenbuterol, Clen had a better rating when it came to anabolic effects. So it would be logical to think that maybe Albuterol has an even higher anabolic effect than clenbuterol due to the shorter half-life, but it isn’t always that simple. There is still much more that needs to be studied and looked at before determining this.

The anti-catabolic effects of Clenbuterol and Albuterol are a plus, but the primary goal of taking either drug is for fat loss. When honestly looking at things, there are far too many other substances that carry more weight in preserving muscle mass than any beta-2 agonist when there is a deficit in calories. Anabolic steroids and human growth hormone cast a shadow over Clenbuterol or Albuterol when it comes to preserving lean muscle mass. Most bodybuilders that are dieting are usually taking one or both of these substances, so the anti-catabolic effect is not much of a factor compared to the fat loss that occurs from either of these drugs.

When comparing Clenbuterol and Albuterol in the category of fat loss, the half-life has to be looked at. Clenbuterol has a half-life of about 48 hours, while Albuterol has a half-life of just about 6 hours. There is an upside and downside to the half-life to both of these drugs. The long half-life associated with Clenbuterol will allow a person to dose less often with the drug still staying active in the system and doing its job with burning body-fat. The downside to this long half-life is that the body stops responding to this drug fairly quickly. This requires a person to cycle the drug 2 weeks on and 2 weeks off, or the person will have to dramatically increase the dose after 2 weeks to feel any effect at all. The short half-life of Albuterol requires a person to have to dose more often throughout the day for the drug to be most effective, but tolerance is not built up nearly as fast as it is with Clenbuterol.

Albuterol has a more mild stimulant effect than Cenbuterol, and some people find it easier to focus while taking albuterol than Clenbuterol. Clen leaves some people shaky, sweaty, and unable to focus during a workout, so albuterol would be the obvious choice. When dieting, some people feel sluggish and find it hard to power though a workout without a strong stimulant. Clenbuterol will often give them that extra edge to push through a workout they normally couldn’t while dieting. Clenbuterol would be the choice for this group.

Both of these drugs fall into the same class of drugs. There are positives and negatives to each one, but when it comes to Clenbuterol and Albuterol, everything kind of evens out at the end of the day. Everyone responds differently to every compound. Some are more sensitive to stimulants than others. I do believe that you will not see much difference in the fat burning and anti-catabolic effects between Clenbuterol and Albuterol.

Always Add Proviron Into Your Cycle

Proviron is an interesting and often overlooked bodybuilding drug. It’s an orally active form of DHT (Dihydrotestosterone) and as such, a Proviron cycle delivers all the good and all the bad that DHT has to offer the bodybuilder.

As for the good, DHT from a Proviron cycle is a pure androgen, but unlike testosterone, DHT doesn’t cause any water retention. DHT is what makes muscle “hard.” And it lowers SHBG (Sex hormone Binding Globulin) which prevents estrogen from forming. By preventing estrogen from forming, DHT can be used to prevent the aromatization of other steroids. This means that DHT from a Proviron cycle can keep steroids in the testosterone family from converting to estrogen and causing in men estrogen related side effects like gynecomastia. DHT also contributes to erectile rigidity. All good stuff! There are drawbacks however, most notably prostate hypertrophy and hair loss. So one may say DHT is pure maleness.

In the past, DHT was regarded as the “bad” form of testosterone and there have been many products available to reduce it. 

So, we know DHT can be tricky and we know we need it. But, where does Proviron fall into the equation? Well, proviron can be a nice addition to any cycle for a variety of reasons. Since it lowers SHBG it allows for more total testosterone to become bioavailable. Up to 90% of testosterone, be it natural or administered, remains in a “bound” state (due to SHBG) rendering it useless for building muscle. Proviron releases more active testosterone into the bloodstream so you can get more of its benefits.

Proviron also works well as an anti estrogen, in fact, it’s superior to most traditional anti e’s such as Nolvadex because there is no rebound effect. Instead of removing estrogen, proviron prevents the formation of estrogen in the first place.

Proviron is excellent for contest preparation because it adds density and definition. You see, androgen receptors are found in your fat cells as well as muscle cells and DHT binds so well that there’s a distinct fat burning effect from its use.

Many people use Proviron in between cycles to maintain muscle gains and libido. However, DHT will not do much in regard to maintaining muscle. And as with any drug, a tolerance is developed over time so using Proviron to help a lagging libido will just add to further suppression once the use of the drug has ceased.

The main downside of Proviron is that since it contains no anabolic properties, it will not result in much muscle growth if used alone. It truly is a “kicker” to other steroids, most notably testosterone. And although it doesn’t cause water retention, it can increase blood pressure. DHT can also increase irritability if duration exceeds more than a few weeks.

There have recently been advances in natural alternatives to Proviron. Avenacosides have been shown to lower SHBG and to raise free testosterone by as much as 20%. Since Proviron is used mostly as an ancillary substance and does not have much of an effect of actual growth, more and more people are opting to go the natural route. Although Avenacosides won’t work quite as well, they are effective and can be used in conjunction with a lower dose of Proviron for an even more powerful muscle hardening/libido enhancing effect.

What is Clenbuterol?

Clenbuterol is a bronchodilator used to threat asthma and other conditions. It belongs to the sympathomimetics group of drugs, which affect sympathetic nervous system, mostly by affecting beta or alpha receptors. In Clenbuterol’s specific case, its effect results on increased fat loss.

How does it works?

Being a beta-2 agonist, Clenbuterol works as a fat burner by binding with beta receptors (mostly type 2 in humans) in fat and muscle tissue in the body. When this bond is formed, the receptors then start a series of chemical reactions leading to the production of cAMP. cAMP then produces and activates enzymes which induce fat breakdown.
Given that Clenbuterol has little effect on beta-1 receptors, it is capable of reducing reversible airway obstruction, with less cardiovascular effects, if compared with nonselective agonists. This whole process will create an increase in the body’s temperature, which will make the body burn more calories. However, your body will attack this matter through beta receptor down regulation, which will dictate how long can you run clenbuterol and still get results. Clenbuterol also lowers lipoprotein lipase activity. This prevents fat deposition and makes fat more responsive to the Hormone Sensitive Lipase activity described above. It is also important to mention, that beta-2 agonists such as Clenbuterol have been shown to increase T3 levels, thus enhacing clenbuterol’s fat burning capabilities.

Another benefit of Clenbuterol, are its anti-catabolic properties. This occurs as Clenbuterol blocks both ca++ dependent proteolysis in rat skeletal muscle, as well as the ubiquitin-proteasome proteolytic pathway. When beta 2 blockers are administered, the effect is not observed. Blocking these pathways inhibits muscle tissue breakdown.

One important thing that must be observed, is that in the past it was believed that Clenbuterol could be used as an Anabolic in humans. This was based on several studies performed on livestock and rats, which in fact showed such results. However, the doses used on these animals, if translated into human doses, would probably kill a person.

How is Clenbuterol dosed

For males the common maintenance dose of Clenbuterol is between 120-140mcgs per day. For females the dose of Clenbuterol goes down to around 80-100mcgs per day. It should however taken seriously, and each person must stablish a maximum safe dose, based on their experience.

How should I cycle Clenbuterol?

This is a very important point of every drug, that some pleople overlook, or just don’t go into enough detail. Clenbuterol should be cycled 2 weeks on/2 weeks off (this can change if ketotifen is used-see below). This is caused by two major factors.

  1. Beta-2 receptor down regulation: this will cause the effectiveness of Clenbuterol to be reduced as time goes by.
  2. Long half life: it has biphasic elimination, with the half-life of the rapid phase being about 10 hours, and the slower phase being several days. This cause the theory of using it 2 days on/2 days off to flaw, as in fact you would be on the whole time, and your beta receptors would not have enough time to recover.

If you use ketotifen you could in theory stay on Clenbuterol for an undefined period of time. This is because contrary to Clenbuterol, ketotifen up regulates beta receptors. The recommended dose of ketotifen is 2-3mg per day, and it should be taken before bed, as it causes drowsiness.

A typical Clenbuterol cycle for a male would be something like this:

  • Day 1: 20mcgs
  • Day 2: 40mcgs
  • Day 3: 60mcgs
  • Day 4: 80mcgs
  • Day 5: 100mcgs
  • Days 6-14: 120mcgs

Please be aware that you must set your own limits, and listen to your body.

What are the possible side effects?

Common side effects of Clenbuterol are increased perspiration, insomnia, muscle spams, restlessness, palpitations, involuntary trenbling of the fingers, nausea and increased blood pressure

Dealing with Injection Pain

Injection is not the most popular method of administration for those new to steroids, but as they will quickly learn, injectables are the safest for men in terms of long term health and by following proper injection instructions, the risks are minimal. However, with this in mind, injections can be difficult as it may result in pain and even infections. In this article we will examine some of the reasons this happens and ways to avoid it.

Causes of Injection Pain
One of the main reasons injections can be painful is the esters involved. Esters are basically buffers that slow the release of a steroid into your system and this allows for less vigorous injection cycles. The pain is caused after the steroid has been released leaving just the ester to crystallize which makes it take longer to dissolve while longer esters dissolve more readily. This is just one reason why it is a good idea to alternate your injection site as you do not want these crystals to build up and cause pain. Additionally, steroids that are released quickly can also cause pain, the worst offender being water based injections or injections with no ester at all. Additionally, injecting too quickly can tear muscle tissue while injecting into a new spot can be painful as the muscle will not absorb the compound as quickly.

Methods of Relief
One method of alleviating pain is to add additional sterile filtered oil to your injections at a ratio of 50:50 and injecting slowly (30 seconds per ml). One may also take some ibuprofen to decrease any swelling. Another strategy is to freeze the tip of the needle thus decreasing the pain as it pierces the skin.

In addition to all of these strategies, it is also important to keep everything you are using clean, to choose the right needles, to pick the right injection sites, and follow the proper injection guidelines accordingly in order to avoid infection. More information on these topics can be found here.

Infection
Infections can be identified by these localized four traits: heat, pain, redness, and swelling. You may also experience chills, fever, and pus excretion. If you believe you may have an infection, you should see your doctor or local hospital immediately. Although it can be a good idea to keep a various array of antibiotics at home, unless you see fast results you may risk giving the infection enough time to enter your bloodstream. Also, the misuse of antibiotics can create immunity in the bacteria making it resistant to treatment and therefore a threat to others. Always talk to a doctor when administering an injection, it could save your life.

Risks/Side Effects of Trenbolone

Trenbolone does not exhibit any estrogenic activity and therefore estrogenic side effects are not a concern with this compound. It is also resistant to the 5 alpha reductase enzyme, but this is of little comfort to a user as trenbolone is already of the most androgenic drugs in common use by steroid users. For this reason androgenic side effects should be expected by most users that undertake a cycle of this drug. Prostate enlargement and oily skin/acne are commonly reported by users. As well anecdotally many users have reported that trenbolone is one of, if not the, harshest compound for losing one’s hair. If a user is genetically predisposed to male pattern baldness he may want to avoid trenbolone.

Having listed the harsh androgenic nature and side effects associated with trenbolone, it should come as no surprise that women are not recommended to use this compound. The usual virilizing effects such as deepening of the voice, body/facial hair growth, and enlargement of the clitoris, among others are likely to cause problems for female users. These effects can appear at even relatively low doses. Trenbolone is not a compound that women should attempt to administer.

Now due to the lack of estrogenic side effects associated with trenbolone it would seem that users would have little to worry about in terms of side effects like gynecomastia, water retention, etc. However trenbolone is a progestin, meaning that it has the ability to bind to receptors of the female sex hormone progesterone. Also, like other 19-nor compounds trenbolone increases prolactin levels. Side effects related to these reactions can include breast growth and lactation. To prevent these side effects as they relate to increased prolactin levels a user can use several compounds including bromocriptine, vitamin b6, and/or cabergoline. Letrozole can also be used to lower progesterone levels. It should also be noted that trenbolone lowers thyroid levels temporarily which in turn raises prolactin levels. It is therefore advisable that users may want to use the compound T3 to combat this effect in part.

Being a progestin, trenbolone also has a dramatic effect on users’ natural testosterone production. Much in the same way that nandrolone does, trenbolone can suppress the natural production of testosterone for weeks after a user has ceased administering it. For this reason it is advisable that users use testosterone in conjunction with trenbolone if they wish to avoid sexual dysfunction, libido problems, or mental side effects associated with a lack of testosterone. Anecdotally many users have also reported that testicular atrophy is nearly always a problem when using trenbolone and that it is much more dramatic than with other compounds. Users may wish to administer human chorionic gonadotropin to help counteract this.

The psychological effects of trenbolone use are also quite distinct in some users. Of course the obvious effect would be a reaction to the androgenic nature of the compound. An increase in aggressiveness is often reported by users, as androgens help to affect brain chemistry and may cause feelings of well-being, angst, aggression, or anxiety. As well, anecdotally some users have also reported that they experience vivid dreams while using the compound.

Kidney and liver function is negatively affected by trenbolone use as well. It is recommended that users closely monitor these throughout a cycle making sure that no problems arise. As well, users will also report that darker than normal urine is a common side effect of use of trenbolone. This should not be a cause of alarm among users; however they should ensure that there is no blood in the urine as this is obviously a sign of trouble.

Comparison between Trenbolone Enanthate and Trenbolone Acetate

Trenbolone enanthate unlike testosterone-based steroids, does not result in the development of feminine sexual characteristics in men. This steroid has the ability of stimulating protein synthesis gains that prove useful in new tissue formation that, in turn, promote muscle growth or hypertrophy. Moreover, Trenbolone enanthate is used by those into boxing, MMA, and cycling as it helps with strength and speed. Trenbolone enanthate can demonstrate unmatched efficacy in reducing body fat levels as it has a strong cortisol-reducing effect along with the ability to bind to the glucocorticoid receptor.

Trenbolone Acetate

Trenbolone Acetate has the molecular formula of C20H24O3 and has an anabolic-androgenic ratio of 500:500. It has an active life of 2-3 days and has the molecular weight of 270.3706 g/mol at the base. Trenbolone Acetate, a 19-nor steroid, is derived from the compound Nandrolone. It is characterized by strong androgenic properties and no estrogenic activity. Trenbolone Acetate has a binding affinity for androgen receptor five times the strength Testosterone. Trenbolone acetate is admired globally by elite athletes as it can dramatically improve the uptake of nitrogen by muscles and improve the level of protein synthesis. Ideal for cutting cycles, Trenbolone Acetate does not get metabolized by aromatase or 5α-reductase into estrogenic compounds like estradiol or into Dihydrotestosterone (DHT).

The primary difference between Trenbolone Acetate and Trenbolone enanthate is esters. While Trenbolone enanthate, featured by comparatively less esters, peaks at a slow pace and leaves the system slowly, Trenbolone Acetate with more esters peaks faster and leaves the system faster. Moreover, Trenbolone Acetate is better when it comes to maintaining stable blood levels, especially when athletes want to gain muscle mass and strength when following a dieting regimen. Furthermore, the human body finds it simpler to absorb a higher percentage of milligrams when injected with the acetate form rather than in the enanthate form.

A big majority of athletes and bodybuilders, especially those into elite sports and professional bodybuilding, prefer Trenbolone acetate over Trenbolone enanthate. This is because the enanthate version of Trenbolone doesn’t result in as much hardening of muscles as experienced with the acetate version. While the acetate version is known to promote cutting, the enanthate version is used for adding muscle size. Beginners to the world of anabolic compounds should opt for Trenbolone Acetate instead of Tren E as Tren acetate gets out of the system quickly. However, the enanthate version of Tren is rarely associated with night sweats, insomnia , and over-aggression and allows athletes to reap the optimum benefits of the potent steroids. Trenbolone enanthate is less likely to result in the dreaded Tren cough and involves less pinning but the use of this steroid involves more of patience as results can take time.

The fact that Trenbolone acetate is easily and more readily available puts it ahead of the enanthate version. In addition, this short ester compound is better than enanthate version when it comes to controlling and manipulating blood levels.

Trenbolone acetate : beast in a bottle !

When it comes time to choose which steroids to run the athlete is left with a host of choices likened to that of a fat kid picking out his first piece of candy; the choices are endless! But what’s best? That’s the question so many are often left with; what’s best for putting on size, what’s best for dieting, what’s best for strength; simply what is best? Then there are the questions of safety and side-effects and these can vary from person to person because like all things in life, various steroids can affect individuals quite differently. Putting that aside, assuming we are all the same for arguments sake, when it comes to the world of anabolic steroids we are left with one; there can only be one and that one is Trenbolone Acetate (Tren.) If you’re looking for a thorough profile of exactly how the drug works there are tons of places online you can go; and in our articles section and their steroid profiles for some very good specifications. Now we’ll touch on some of those here but our main concern with this overview is simple, explaining why tren is the king of kings.

Trenbolone is a 19 nor steroid, simply meaning the testosterone molecule has been changes in the 19th position and low-and-behold, you have Trenbolone. Sure, we could go into more specific detail but for most bodybuilders the specifics do not matter, only that it works and works well; again,

One of the most potent agents on the market, tren has been said to be 400% to even 500% more powerful than testosterone; that alone should make your mouth water! Further, unlike so many anabolic sterods, water retention with tren is in many cases non-existent with nearly all the gains being pure 100% muscle tissue. If you’re unfamiliar with Trenbolone, the above should be enough to have you chomping at the bit but it gets better, much better. When it comes to training, the name of the game is recovery; growth, in terms of size or strength and even in shaping in prepping for a contest, it is in recovery that progress is made. It is the training in-which ignites the fire but the recovery that molds the molten metal. It has been shown that tren has the ability to increase muscle-cell repair by nearly 100% greater than repair without tren. What does this mean in simple terms? It would mean your cells and fibers are repaired twice as fast. How accurate is this? Hard to say if it is pin-pointed to that degree exactly but it’s not far off. The reason for this recovery is tren’s ability to greatly increase the production of the hormone IGF-1

Trenbolone has been shown to greatly aide in fat loss . It’s no secret, bodybuilders love tren for contest prep, the hardness is brings is unlike any other; its potency allows them to hold to maximal strength as long as possible throughout their prep but the addition of fat loss benefits are simply an added bonus. Now couple this with a growth season, someone looking to add size and strength, this makes tren a perfect choice, even more so for the off-season bodybuilder. Think about it, it’s quite simple; Trenbolone aides in increasing size and strength, yes, you still have to eat to fuel those gains but now the gains of excess body fat becomes less of a concern; I’m sorry, if you’re not loving tren already you have a problem.

As mentioned, as said a million times, you have to feed your muscles, it’s that simple. What if there was a compound available that would allow you to gain more from the same amount of calories you’re eating now? What if there was a compound in-which enabled your body to absorb the nutrients more efficiently? For example, take two bodybuilders, BB “A” & BB “B” both eat a diet of 3,000kcl per day, both are genetically identical and eat identical diets. Both are using anabolic steroids; both are running testosterone and both decide to stack it with another compound. BB “A” chooses Trenbolone; he has now created a means for his body to make more use of each nutrient in his diet. BB “B” chooses Anadrol, a very potent steroid in its own right; he will make great gains too. However, BB “B” will soon realize much gained has been fluid, yes he gained lean tissue as well but he did not enjoy the boost of nutritional efficiency BB “A” enjoyed.

Yes, there are absolutely side effects to Trenbolone, here we are simply discussing the benefits.

  • No estrogen conversion
  • Perfect for hardening
  • Perfect levels of nutritional intake absorption
  • Massive strength gains
  • Incredible cell repair abilities
  • Aides in fat loss
  • Bonus (Not mentioned above) Trenbolone destroys cortisol the evilest of hormones that destroys muscle tissue and can bring a world of havoc on the hard dieter.

There is nothing wrong with Trenbolone Enanthate (Tren-e) or Trenbolone Hexahydrobenzylcarbonate (Tren-hex or Parabolin) nothing wrong at all and either of these is better than no tren at all. However, to begin, tren-a is easier to maintain stable blood levels with and this is very important, especially when dieting and yes even when trying to gain. Further, milligram for milligram, studies have shown the body has an easier time absorbing a higher percentage of milligrams when injected in the Acetate form rather than one of its cousins.

This should give you a good understanding of tren and how perfect of a compound it is when you’re choosing which anabolic steroid to run. In most cases tren should not be run by itself; tren will shut down your natural testosterone production and you’re best suited to stack tren with some form of testosterone. Also, an important note, Trenbolone is typically not suitable for beginners; Trenbolone as we can see is a very, very potent compound and the side effects in some cases can be brutal for some lifters. That is also another reason why Trenbolone Acetate is best in comparison to other forms of Trenbolone. Because it has the Acetate ester attached to it, making it very fast acting and giving it a very short half-life, if problems arise, the lifter can discontinue Trenbolone Acetate and have it cleared from his system very quickly.

Trenbolone and women

Although women are generally told to avoid using this drug, Trenbolone is being used more and more by women in controlled doses.

Trenbolone is one of the most popular drugs used by bodybuilders today and, when you look at the stats, it is not hard to see why. A very potent androgen with strong anabolic activity, Trenbolone is an extremely effective hardening and cutting agent. In fact, it is considered indispensable when it comes to pre contest preparation. However, it is also extremely valuable in the off-season as it creates a rapid build up of strength and muscle mass. In fact, the anabolic effect is often compared to Testosterone or Dianabol with one very important difference 1/2 it does not convert to estrogen. This is what truly sets it apart, as most mass building drugs readily aromatize, leading to many estrogen related problems (e.g. water retention gynecomastia).

Due to the lack of water retention, the gains when using this drug are more easily maintained on discontinuing its use. In addition to this, a very hard and defined appearance can be achieved. Also, since gynecomastia is not an issue, there should not be any need to add an anti-estrogen as long as Trenbolone is the only steroid being used. Due to the highly androgenic nature of this drug an increase in the burning of body fat is observed and a much tighter physique can be achieved without having to resort to extreme dieting.

Trenbolone is more potent than testosterone with an effect being gauged as three times as strong on a milligram for milligram basis. It is also four times as anabolic as Deca Durabolin and ten times as androgenic. This makes the majority of the weight gained on this drug lean, quality muscle. trenbolone also creates an increase in the levels of the hormone IGF-1 (insulin like Growth Factor-1) which is highly anabolic within muscle tissue. trenbolone has a stronger binding affinity to the androgen receptor than testosterone. This feature is a major contributing factor to the process of anabolism and fat loss. By promoting nitrogen retention and Protein synthesis within the muscle trenbolone allows the food you eat and the nutritional supplements you take to be used more effectively. It also reduces levels of the catabolic hormone cortisol. Trenbolone is also involved in the production of red blood cells and increases the rate of glycogen replenishment (both of which contribute not only to stamina but also to recovery from workouts)

A reduction in aerobic capacity is the most common complaint with Trenbolone. This is thought to be caused by bronchial dilation resulting from an increase in prostaglandin production. The condition known as “tren Cough” is often a complaint registered with users of the Acetate version (trenbolone is available in Acetate and Enanthate forms). Androgenic side effects may also be experienced which include oily skin, aggressive behavior, and acne and hair loss. For this reason women are usually advised to stay away from this drug.

It seems that although women are generally told to avoid using this drug, trenbolone is being used more and more by women in controlled doses. The fact that it adds primarily lean mass whilst reducing body fat is obviously a key factor in its attractiveness. When women were asked for their feedback on trenbolone use a variety of favored dosages came up. Anything from a very conservative 10mg every other day to a more adventurous 100mg/week split into two doses. Stacking Trenbolone with Testosterone Propionate was also something favored by those engaging in high level competition. Another use of Trenbolone involved taking it 3-4 days before a show in order to add hardness and definition to the physique.

It has to be said that side effects were experienced by all - usually increased hair growth and acne - and the severity of the side effects seemed to be worse in younger women. The theory expressed here being that ovarian function may be the reason for this, with a younger woman still having stronger ovarian function than an older women who may be entering peri-menopause. This is all speculation of course but seems like a plausible explanation in my opinion. Either way, if you are considering using trenbolone it is advised to use it on its own and at extremely low doses (such as the aforementioned 10mg every other day) in order to test your own unique sensitivity.

trenbolone is a potent androgen that is primarily used in cattle, so there is even less information at our disposal on this compound or its effects on the female endocrine system than any other drug. It is the one drug that seems to produce results as significant as the side effects that are associated with it. Women are generally advised to stay clear of Trenbolone considering the strong androgenic component which eradicates any possibility of running tTrenbolone without sides. The more seasoned female athlete will run it in the off season in order to reap the muscle building benefits of the drug whilst maintaining a relatively low body fat. On the other hand running it during contest preparation will preserve the newly added muscle mass while on a calorie restricted diet. The less daring athlete will run Trenbolone during the last few weeks of contest preparation or even limit their use to the week before the show - with a more frequent injection schedule.

Women who have experienced less favorable side effects on Trenbolone report experiencing tachycardia from a single pin, accompanied by profuse night sweats and insomnia bad enough to bail on the cycle. Others experience rapid hair growth with more frequent shaving (side effects that are far from unmanageable). Quite honestly, Trenbolone dosing is dependent on how much a woman is willing to deal with in terms of sides. There is no conservative dose for a first timer with trenbolone being far better suited for the educated, experienced and seasoned athlete who has paid her dues.

Training on steroid cycle. What Supplements to take and how To Eat and Diet.

Another very often asked question is:

"How should I eat and lift when on cycle"

The answer is that your diet and training should be about the same on or off with some minor tweaks. What I mean is that you should already have a good plan in place for diet and training…if not you shouldn’t be doing gear.

The only differences should be that you can work out longer because you will recover faster between sets, you will be able to lift heavier because of the increased strength, your workouts can be more intense from increased agression, and you can do it more often because of improved recovery. I can easily do 2 a day workouts on gear but my training is pretty similar to my training otherwise, I just condense the timeline and increase the volume.

Instead of working a muscle group once every six days you can now do it once every 3 days. If you were doing full body training 3 times a week you can now do it 6 times a week. Overtraining is a lot harder to do on gear but you need to make sure you are eating and sleeping enough that the gear can do it’s job.

This seems to really help bodybuilding type programs when you are doing a crap load of sets. It will also help a powerlifting program where you are doing heavy compound movements because you will recover better between sets and have more explosive power. Whether you’re doing West Side, German Volume Training, or Full Body, or whatever the important thing is that you really focus in the gym (even the best program sucks if you don’t put your best effort into it). Whatever you are doing it will work as long as you are doing it properly. Add weight, add reps, add sets and you get stronger because that is the body’s only choice. Whatever you do - do it balls to the wall and you will see results.

The only word of caution is to not drop below the 4 rep mark for sets that you can perform with good form. I have heard of many a guy on juice tear a biceps or fuck up a disk or something because he was being a hero and going for 1 rep PRs. There is a strong desire to do so because you are so much stronger you will want to know what your “geared PR” is but resist the urge, it’s masterbation for your ego. The only exception would be if you are a powerlifter or someone that is used to training in the very low rep range all the time (ie your ligaments have adapted to that type of training over a long period of time).

The problem is that your muscles are rapidly getting stronger because of the gear but your ligaments are not…this is especially true with drugs like Deca Durabolin, Trenbolone, and Winstrol that add a lot of strength very fast without giving the body time to adjust. Some drugs will also give a false sense of security because the joints feel great (well lubricated and loose) or actually help repair old injuries (Deca Durabolin is good for this because of it’s effect on cartilage which was covered earlier) but this doesn’t mean they are indistructable so use your judgement. You will make very good gains sticking to the 4-12 rep range…work with that.

Diet depends on your goals but basically:

  1. You can bulk steroids better/cleaner  - Your body can make use of more calories on cycle because your body is in a state of accelerated protein sysnthesis and your body on gear will make better use of calories you put in even if they are a little sloppy (nutrient partitioning will be improved such that more cals will go to your muscles).
  2. You can diet better - You can also maintain more muscle mass in a fasted state so dieting hard will not have the muscle wasting effect that it normally would.

For bulking or gaining you should be taking in at least 1,000 extra calories a day and upping your protein intake so that you are getting a MINIMUM of 1gm/lb of body weight (many pros recommend upwards of 2gm/lb), that’s a lot of protein but it is needed to really see results. I think eating clean is still a good idea but it is crutial to eat enough to grow. A lot of people who have trouble gaining don’t eat enough. If this is a problem without gear it will be a problem when on the gear. Figure out what you need and make sure you have an idea of what that actually entails in terms of a daily food breakdown. Plan out your meals and see what the total Protein, Fat, and Carb breakdown is. It is often much less than you think it is.

The flip side for dieting is that you can also get away with eating less and not losing muscle mass like you would naturally. This means cutting at least 500cals/d while still maintaining the minimum 1gm/lb of bodyweight for protein. Make sure you are getting healthy fats so that you can absorb all the necessary fat soluble vitamins and if you are restricting carbs make sure you have some in your system while training and right after so that you have energy to train and you are replenishing glycogen stores after.

The same general rules apply for eating - 5-7 small meals throughout the day. Don’t go hungry or go to sleep on an empty stomach. eat slow digesting foods like protein, healthy fats, and whole grains. don’t eat crap (fast food, fried stuff, processed grains, sweets) you can gain weight without this stuff (or very little of it if you are very lean naturally), the guys that say you need to eat like that on gear are usually either taking so much gear that they could eat scrap metal and gain or are big fat guys or both.

Typical “clean foods” are:

  • Lean Meats/seafood (chicken, lean beef, bison, venison, moose, horse, shrimp, scallops, tuna, salmon, or any other fish as they are all fairly healthy)…this should make up the bulk of your calories so that protein intake is high enough to get the daily minimum.
  • Eggs and Dairy (whole eggs, egg whites, milk, low fat cheeses, yogurt), for those that can tolerate dairy it is a good addition to get some extra protein. Remember that milk and yogurt have a fair amount of carbs so if restricting carbs it will need to be reduced or eliminated.
  • Vegetables (especially leafy green ones which are higher in fiber and anti-oxidants), if you are restricting carbs then tomatos, peppers, potatos, and yams should be reduced but otherwise veggies should all be fair game. veggies are fairly filling but low desity calorie wise so if you are having trouble eating enough while bulking it may help to restrict veggies a bit.
  • Healthy Fats (olive oil, avocado, nuts, natural peanut butter, macadamia nut oil, fish oil, milled flax seeds), the body needs healthy fats to absorb fat soluble vitamins and keep LDL/HDL ratio in check so make sure you are getting some.
  • Fruit (pure fruit juices and whole fruits like oranges, berries, bananas, pineapple, etc)…fruits are a great source of anti-oxidents and a good source of carbs. If you are restricting carbs then you need to avoid fruits and definitely avoid fruit juices.
  • Whole Grains (oatmeal, brown rice, wild rice, whole wheat pasta, whole wheat bread), obviously these need to be eliminated if cutting out carbs. Carbs are very anabolic food so they are definitely needed if bulking.
  • Beverages (water, geen tea, coffee, diet soda) these are about the only drinks that don’t contain a ton of sugar. The sugar used in drinks is often glucose fructose which is thought by many to be one of the root causes of the obesity/diabetes problem so it goes without saying it should be avoided.

Supplements

The following supplements are ones that I think are good and work for me. Different ones may work for you so please take everything with a grain of salt.

  • Protein Powder - This is about the only way I can manage to get my daily protein requirement in because meat is expensive and it takes longer to digest so eating a few pounds of it a day is tough. obviously it is important to choose a good one so you aren’t just taking in a whole ton or sugar and crap. Biotest’s is good. It’s not the only one I like but it is what I drink most often. Things to look for is: fast and slow digesting proteins, good quality of protein, and fairly low in carbs so you aren’t just getting a glass of sugar.
  • Fish Oil - good for a lot of reasons: joints, lipids (LDL/HDL ratio), cardiovasclar health, reduces inflamation, immune function, etc…Omega-3’s are very good for you and fish oil is very high in it.
  • Creatine - this is one of the most popular of all time supplements and works well. It’s now considered safe to take year round just make sure you drink lots of water with it.
  • BCAAs - I have finally jumped on the BCAA band wagon. I find that I perform a lot better in the gym and on the field with them (I usually take about 10 before training and find my energy level is much better). If it is sparing muscle in the process then that’s great but the number one thing for me is that my workouts are better on it.

Trenbolone Side Effects: Night Sweats and Reduced Cardio Capacity

Q: “My trenbolone cycle has greatly reduced by cardio capacity, and the night sweats are extreme. Yet two friends are doing the exact same cycle as me with no issues at all. Is this always going to happen to me with trenbolone? Even with these problems, the results are worth it.”

A: Both reduced cardio and night sweats are somewhat common complaints with trenbolone users, though in the majority of cases neither occurs. The cardiovascular capacity problem is in at least some cases related to elevated hematocrit. It’s worth checking. At the highest level of competitive cycling, it’s well known that athletes have sought high hematocrit levels. With high hematocrit, they’ve been able to sustain increased power output. Or in other words, their cardio performance was better. However, this isn’t a general rule. Along with higher oxygen carrying capacity, along with higher hematocrit comes higher blood viscosity, or greater blood thickness. Even where hematocrit is remaining within the normal range, one study found that aerobic capacity of other athletes may be superior with midrange or even low-midrange hematocrit than with high-normal values. It’s possible for hematocrit to go substantially above normal during an anabolic steroid cycle. Since you are seeing an adverse side effect, I’d check hematocrit level. If it is at about 53 or higher, I’d discontinue anabolic steroid use for now. If hematocrit is in the normal range, then there’s no known answer for the reduced cardio ability. Some have speculated that lung irritation might be the cause. I don’t know of medical evidence, but don’t rule out that there could be truth to it. To deal with the cardio issue, some trenbolone users limit their dose to for example 50 mg/day rather than 75 or 100 mg/day. Trenbolone is so effective per milligram that even 50 mg/day provides an excellent effect as part of a steroid stack. As for night sweats, I know of no convincing explanation for this problem. It seems to strike randomly. Commonly, for the same individual it will occur in some cycles but not others. There seems no solution but getting the bedroom as cool as possible and using beach towels to absorb the sweat. At least the night sweats may be helping to burn off fat.

Clomiphene citrate (Clomid) by Geneza Pharmaceuticals

Clomiphene citrate is an anti-estrogenic drug that is prescribed to women to treat anovulatory infertility (inability to ovulate). In clinical medicine it is specifically referred to as a nonsteroidal ovulatory stimulant.

The drug works by interacting with estrogen receptors, often in an antagonistic manner, in various tissues of the body including the hypothalamus, pituitary, ovary, endometrium, vagina, and cervix. One main focus is that the drug will oppose the negative feedback of estrogens on the hypothalamic-pituitary-ovarian axis, enhancing the release of gonadotropins (LH and FSH). This surge in gonadotropins may cause egg release (follicular rupture), ideally leading to conception.

Clomiphene citrate is chemically a synthetic estrogen with both agoniist/antagonist properties, and in this regard is very similar in structure and action to Nolvadex®. It is believed that both the estrogenic and anti-estrogenic properties of clomiphene citrate play a role in its ability to support femal,e fertility. In men, clomiphene citrate also acts as a partial antiestrogen, and may be used to counter some of the side effects of aromatizable steroid use including gynecomastia and increased water retention.

As an anti-estrogenic drug, clomiphene citrate may also produce an elevation of follicle stimulating hormone, and luteinizing hormone levels, which can elevate testosterone production. This effect is especially beneficial at the conclusion of a steroid cycle, when endogenous testosterone levels are depressed. Here, clomiphene citrate is most often applied in combination with HCG and tamoxifen, in an effort to restore endogenous testosterone production more quickly. If testosterone levels are not brought back to normal in a short period of time, a significant loss in size and strength may occur.

This is due to the fact that without testosterone (or other anabolic/androgenic steroids) to impart an ongoing anabolic message, the catabolic hormone cortisol becomes the dominant force affecting muscle protein synthesis. Often referred to as the post-steroid crash, when not corrected this state of imbalance in the endocrine system can quickly reduce muscle mass levels, diminishing the long-term return on anabolic/androgenic steroid therapy.

Note that the triphenylethylene compounds (toremifene citrate, tamoxifen citrate, clomiphene citrate) tend to be somewhat intrinsically estrogenic in the liver. This means that while they can block estrogenic activity in some areas of the body, they can actually act as estrogens in this other key area.

Estrogenic action in the liver is important in the regulation of serum cholesterol (it tends to support HDL synthesis and LDL reductions). Since steroid-using bodybuilders are already dealing with the negative cardiovascular effects of these drugs, compounding the issue with aromatase inhibitors (which will lower total serum estrogen levels) may not always be the best option.

Using a drug that blocks gynecomastia, for example, while at the same time supporting improved cholesterol values, might be much more ideal.

How Supplied:

Clomiphene citrate is most commonly supplied in tablets of 50 mg.

Structural Characteristics:

Clomiphene citrate is classified as a selective estrogen receptor modulator, with both agonist and antagonist properties. It has the chemical designation 2-[4-(2 chloro1, 2- diphenylvinyl) phenoxy] triethylamine dihydrogen citrate.

Some patients using clomiphene citrate notice blurring or other visual disturbances such as spots or flashes. These symptoms occur more frequently at higher doses or longer durations of therapy, and often disappear within a few days or weeks of use. Prolonged visual disturbances have been reported after the discontinuation of clomiphene citrate therapy, however, and in some cases may be irreversible. Those taking clomiphene citrate should be warned that these symptoms might make activities like driving a car or operating heavy machinery more hazardous than usual.While the exact cause of these visual symptoms is not yet understood, it is advisable to
discontinue treatment and have a thorough medical/opthalmological examination should they occur.

Side Effects:

Clomiphene citrate appears to be well tolerated, with a low incidence of significant side effects. Common adverse reactions during clinical trails included ovarian enlargement (13.6%), vasomotor flushes (10.4%), abdominal discomfort (5.5%), nausea/vomiting (2.2%), breast discomfort (2.1 %), visual symptoms (1.50/0), headache (1.30/0), and abnormal uterine bleeding (1.30/0).

Data also suggests that the prolonged use of clomiphene citrate may increase the chance of ovarian tumor. Clomiphene citrate is occasionally associated with a serious and potentially life threatening side effect called ovarian hyperstimulation syndrome (OHSS). Early warning
signs of OHSS include abdominal pain and distention, nausea, diarrhea, and weight gain.

Administration:
Clomiphene citrate is FDA approved for the treatment of women with ovulatory dysfunction preventing pregnancy.

The recommended dosage is 50 mg daily for 5 days, which is initiated approximately 5 days into the menstrual cycle. If ovulation does not occur, follow up cycles may use a dosage of 100 mg per day for 5 days.

Many clinicians recommend a limit of 6 courses of therapy. When used by men (off-label) to mitigate the estrogenic side effects of anabolic/androgenic steroid use, a daily dosage of 50-100 mg (1-2 tablets) is usually administered while any offending steroids are taken.

Note, however, that tamoxifen is usually given preference over clomiphene citrate for this purpose. More commonly, clomiphene citrate is used by men at a dosage of 50-100 mg per day for 30 days at the conclusion of a steroid cycle, in an effort to bring natural testosterone production back to normal levels. Here, it is usually deemed most appropriate to use as part of a multi-component post-cycle recovery program. Female athletes occasionally use clomiphene citrate for the reduction of estrogenicity near the time of a bodybuilding contest. In some instances this may aid in increasing fat loss and muscularity, particularly in female trouble areas such as the hips and thighs. The drug, however, often produces very troubling side effects in pre menopausal women, and is likewise not in very high demand among this group.

Bodybuilding and Steroids

Lance Armstrong’s public castigation prior to having his Tour de France titles stripped, and graver punishment of confessing to a disapproving Oprah Winfrey of his doping days and winning ways, revealed one of his self-justifications. He researched the ethics of violating doping regulations, and decided after consulting a dictionary that he was not cheating, as he was not gaining any advantage over his competitors.

Resorting to testosterone, EPO, other drugs as well as blood doping was a perceived need for Armstrong, who said, “I viewed it as a level playing field”1… the everyone else is doing it defense that stopped working for most of us in kindergarten.

Bodybuilding (other than drug-tested organizations) suffers from the same “level playing field” mentality as cycling, without the apparently ineffective barrier of rules regarding performance-enhancing drug use. At the level of the Mr. Olympia, the perfect combination of genetics, training, diet and drugs is required to excel. The number of contestants has escalated (some of the early Mr. Olympia contests had a single competitor, while today over 30 qualify and approximately 20 compete onstage), and the margin between winning and fading in the ranks narrows every year. It is a given that the vast majority of competitors use anabolic steroids dosed far into the supraphysiologic range (generally between 2,000 to 5,000 mg total androgen intake weekly). I would contend that the use of anabolic steroids is universal in this population, but acknowledge the slim (naive) possibility that a truly gifted individual might reach these heights unaided.

By the time a bodybuilder acquires his pro card, he has already committed years of training, financial cost and strained relationships. Entering the pro arena, he arrives at a new tier of competition— much like NFL rookies who excelled in college, only to falter at the pro level. The rewards (e.g., fame, prize money, guest posing, endorsements and business opportunities) are tremendous; the level of risk these men accept encourages them to experiment with potentially life-threatening concentrations of potent drugs and hormones, extreme physical stress and adverse conditions. In the quest to find an advantage, many are turning to unproven experimental drugs and biologics, even surgeries. Every time a new standard for definition or size is set, the bar is raised for all competitors. There are rumors of gene therapy or stem cell implants in elite athletes, making it possible that bodybuilders are also submitting to extremely risky and unethical procedures. This is the irresistible “cookie jar” that has arisen from advances in cellular, genomic and metabolomic science.

There are certain anabolic steroids/hormones that are well established in bodybuilding: insulin, growth hormone and IGF-1 are familiar to many. At the professional level, these drugs have become nearly as ubiquitous as anabolic steroids. Some might suggest the beta-2 adrenergic drug Clenbuterol as another example, but at the doses used in humans, it fails to promote anabolism reliably over the long term. Clenbuterol, and same-class drugs, are used to promote fat loss

Insulin is best known as a glucose (sugar) partitioning hormone secreted from the pancreas in response to a meal, prescribed to treat type 1 diabetes. However, in addition to driving glucose into most tissue (e.g., skeletal muscle), it also activates the uptake of amino acids and stops the breakdown of cellular protein. Insulin also promotes the storage of fatty acids as triglycerides in fat cells. In terms of whole-body growth, insulin is a more potent anabolic than testosterone. Bodybuilders have learned to time insulin injections in conjunction with training and some meals to drive nutrients into recently worked muscle, resulting in greater lean mass gains. The downside is that a miscalculation when preparing an injection or reconstituting a vial could lead to a lethal (life-threatening or life-ending) event. If insulin is overdosed, it can drive circulating glucose (blood sugar) so low that the brain is starved. Unless corrected very rapidly, it can lead to death. Insulin can also increase appetite and resistance to fat loss. It is unclear as to whether insulin or growth hormone first gained widespread use among mass-seeking bodybuilders; combined, they have increased the size and mass of the contestants noticeably. Human growth hormone (HGH) induces a state of insulin resistance which insulin treatments overcome; insulin increases fat storage, which HGH combats.

Human growth hormone was rarely used prior to the late 1980s due to cost and scarcity. Prior to the advent of recombinant technology, HGH was sourced from pituitary extracts of cadavers (the brains of dead people). There are rumors of bodybuilders in the late 1970s or early 1980s using GH derived from the pituitary of rhesus monkeys, but these are not verified by any credible source. HGH has both anabolic and lipolytic (fat releasing) effects, making it very appealing to bodybuilders; the mass gains do not involve the contractile proteins in muscle.3 Thus, increases in size are not associated with increases in strength. HGH use may increase the IGF-1 response to exercise in muscle, but that will be discussed later.4

The hormone also aids in tissue repair, possibly accelerating recovery from tendon or ligament strains. Dosing is an issue with this hormone, just as it is with insulin. Unlike insulin, there does not appear to be an acute toxicity (e.g., hypoglycemia). However, chronic (long term) use of excessive doses of HGH can result in life-shortening conditions (e.g., cardiomegaly, type 2 diabetes, tumor growth) and permanent disfigurement (e.g., overgrowth of facial, hand and foot bones). Over time, bodybuilders have learned to lessen HGH dosing and add the related hormone, IGF-1. The lipolytic (fat reducing) effect of HGH is accomplished at a low dose, whereas the anabolic (net) effect requires concentrations approximately three times greater than physiologic in young men; bringing with the mass gains several adverse effects.

IGF-1 is produced primarily in the liver and skeletal muscle. It is considered a secondary messenger of GH, as it carries out some of the GH-related effects. However, IGF-1 has unique effects and is more specific to anabolic effects in muscle. IGF-1 is produced locally by exercised muscle, and has a role in increasing contractile protein and tendon strength, increasing the hypertrophic response to weight training. Combined use of HGH and IGF-1 allows bodybuilders to experience the benefits of both hormones to a greater degree than relying upon injected hGH and the systemic (liver) production of IGF-1.

If one looks at the bodybuilders of the 1990s and early 2000s, one frequently sees facial deformities, elongated hands and feet and “GH belly” on the larger contestants. Today’s competitors achieve equal or greater size with a lower incidence of these deformities.

However, over time (years), even replacement therapy can cause both physical and metabolic signs associated with acromegaly (GH excess). The point may be moot if the trend of shorter life spans among professional bodybuilders continues.

Other growth factors exist, though are not in use clinically— insulin, HGH and IGF-1 all have established clinical use in medicine. Tissue growth factors are being studied in research labs for use in treating human conditions, or to better understand cellular and molecular processes. Though access to material that other bodybuilders may not have is very appealing to competitive individuals, the risks associated with self-experimentation are immense. These materials often have not been evaluated relative to toxicity, or effectiveness in humans. Further, there are often negative side effects that are unanticipated that might lead to organ damage, organ failure, or death. Even in carefully controlled clinical trials performed by pharmaceutical companies, there are rare cases of severe harm or death. Certain of these growth factors are fairly well understood, and appear to be capable of being used effectively with a reasonable expectation of short-to intermediate-term safety. Long-term safety is a complete crapshoot with absolutely no available data to formulate a guess, especially at the doses being used by these men in conjunction with numerous other drugs.

IGF growth factors are a family of protein hormones that regulate tissue growth and repair. In skeletal muscle, they are prominently involved with exercise-induced hypertrophy and satellite cell recruitment. Among the muscle-derived IGFs are: IGF-IEa, IGF-IEb or IGF-IEc (also known as mechano growth factor, MGF) and IGF-II. These growth factors act locally; circulating IGF-1 (blood concentrations) is often unchanged during this time. MGF acts locally, and promotes muscle growth in ways unique from IGF-1, including activating satellite cells. It would seem that MGF injections would work best administered the day a muscle group is trained, separated from the workout by a few hours. MGF has been detected in black-market products, and it is being explored for treating aging-related muscle loss, supporting the speculation that it may be effective (and in use) as an anabolic agent in bodybuilding.

Shortly after scientists demonstrated the potential to introduce genes into a living host, the concept of “gene doping” was born. As opposed to traditional doping, where hormones are injected into the body at concentrations much higher than normally present, gene doping is a method where strands of DNA are introduced that generate the same hormones at supraphysiologic concentration within the body. It is unnatural because it is not the “blueprint” the person was born with, though it could be argued as “natural” as the resulting hormone is produced entirely within the body and is not injected or swallowed. The concern of anti-doping agencies is that as the hormone is produced in the body, there would be no means of proving that it was not natural. Do not be surprised if we begin to see entire teams of athletes at the Olympics that have mutations or gene copies that are mathematically improbable.

For the bodybuilder, imagine the possibilities- after being infused with tissue implants of satellite cells, Leydig cells, islet cells, brown fat, etc. Then the new genes are activated, issuing concentrations of growth factors, betaadrenergic neurotransmitters, HGH, IGF-1, etc. The result would be nearly limitless growth. As utopian as that sounds, it is the cheese in the trap. Gene transfer is not perfected, and even in the best-case scenario, these growth factors are involved with many disease processes as well (i.e., cancer, enlarged heart, autoimmune diseases).

An area that is anabolic in that it increases muscle size, and is disappointingly prevalent in bodybuilding, is the use of site-enhancing oils such as synthol. The practice of injecting oils into tissue to increase size or (supposedly) cosmetic appeal is decades old. One of the original oils was paraffin oil, once used to augment breast size. Paraffin oil, and later other blended oils, have been used by bodybuilders to increase the size of lagging body parts or to exaggerate dominant muscle groups. Aficionados of bodybuilding are familiar with unsightly lumps in deltoids, biceps, gastrocnemius and other sites. What is not appreciated by those who depend upon these oils is that the long-term effect is destruction of the very muscle being “inflated.” Several cases are present in the medical literature of men who have experienced chronic, severe pain and loss of strength. The damage, detected by MRI or ultrasound, shows pockets of oil, inflamed and scarred muscle. The reported cases resulted in surgical removal of sections of the involved muscle, and segments removed in the surgeries reported were roughly five by two by two inches.

There are other agents with anabolic effects capable of increasing muscle size, such as myostatin inhibitors and specific prostaglandins. It is clear that an ever-expanding cocktail of growth promoters, cellular implants, gene transfer and other techniques are being explored in the eternal quest for greater size/strength. Greater complexity and more numerous concurrent manipulations nearly guarantees foreboding consequences.

While the cavalier may boast it is survival of the fittest, or what does not kill you makes you stronger, the reality is that bodybuilding will become extinct as a consequence of natural selection. Though it is advanced scientific knowledge that is providing the avenues for growth, it is not wisdom guiding the practice as we now see it.

The Trenbolone Experience

Trenbolone is a classified schedule III drug in the United States; which means that it is illegal. It is from the 19-nor family of anabolic steroids just as deca is. The 19-nor stands for the fact that the testosterone molecule was changed at the 19th position to make it into the new and more potent compound. It was originally an anabolic androgic steroid developed and used by veterinarians on livestock to increase muscle growth and appetite and later would make its way to the chemical athlete’s world. The farmers clearly knew that these anabolics were the key to bigger and better breeds, allowing for faster income as well. On the average farmers sell cattle that have less than 28% EBF, they will not exhibit enough finish to reach a USDA quality grade of low choice. The majority of cattle need to have at least 28.5-29.5% EBF in order to grade to their genetic potential.

Therefore, all these factors need to be taken into consideration when choosing an effective implant (Trennobolone acetate) strategy which include feed costs, animal costs, quality grade, genetics, economic advantages of weight (live & carcass), production goals, and carcass goals. There are trade-offs to all the above and implants can help you achieve your goals and benefit you economically in all circumstances.

The recent study noted that when taking Trenbolone with estradiol, the weight gain was more proficient. Of course bodybuilders don’t want to take estradiol, but taking tesostosterone will allow for aromatization which in turn will increase the body’s estradiol production leading to more muscle gain. This one of the reasons why the Test-Tren stack is so popular besides the fact that testosterone helps the impaired libido from Trenbolone.

In many people’s eyes, Trenbolone is pound for pound the strongest compound for pure gains and stacks well with anything. Although some may argue that it’s a “No No”, while on Nanodrolone Decanoate aka Deca. However I have ran both successfully with no more or less problems than running them separate.

Trenbolone significantly increases nutrient efficiency along mineral/vitamin absorption. Feed efficiency is the measurement of how much of an animal diet is converted into meat, and the more consumption of food it takes to finalize this meat, the lower the value. On the other hand, the less food consumed by the animal to finalize the meat; the higher the value/efficiency.


Trenbolone is one of the few anabolic androgenic steroids, that can lead to fat loss without change in diet, and it also has scholarly literature stating so. This makes it perfect on a recomp as you will lose all fat needed but pack on dense muscle at the same time, don’t get me wrong; you can still use it for other goals including bulking and cutting.

Like most all anabolic androgenic steroids the Trenbolone hormone greatly promotes nitrogen retention in the muscles, increases red blood cell count and dramatically reduces and blocks glucocorticoid steroids aka the stress hormone cortisol; this hormone destroys muscle and increases fat storing hormones. This also means that you can do all the cardio you need to lose fat without having to worry about losing your muscle gains. Dieting can also place major stress on the mind and body leading to more cortisol, but Tren can help cease the cortisol release. Tren sounds ideal on a cut if you ask me. Increased nitrogen retention also means increased nutrient deliver to the muscles allowing for more glycogen retention. More glycogen retention leads to better workouts and a fuller look.

As is with nearly all anabolic androgenic steroids the Trenbolone hormone can greatly promote cell repair and regeneration, which will speed up the healing process in the body. The main reason why you experience growth on anabolics is because your recovery times goes up allowing you to become faster stronger. The more weight with frequency you can lift, the more and faster you will grow. One of the reasons why Tren is so good at increasing recovery time is the fact that it significantly increases the body’s natural production of the potent anabolic hormone IGF-1. Tren also causes muscle satellite cells to be more responsive to IGF-1 and other growth factors. The amount of DNA per muscle cell will also experience a significant increase. For these reasons adding Igf-lr3 into a tren cycle is a no brainer.

Trenbolone severely binds to the androgen receptors therefore increasing growth of muscle mass and melting fat away. This is important, because the stronger a steroid binds to the androgen receptor the better that steroid works at activating androgen receptor’s dependant mechanisms of muscle growth. There is also strong supporting evidence that androgens which bind very firmly to the androgen receptor also help in shedding fat. The androgen receptors are like locks and the androgens are like different keys. Some keys open the locks much better than others. This is does not mean that AR-binding is the only way that steroids work. There are anabolics that barely have any considerable binding to the AR and yet display great capability to build muscle and strength. Trenbolone is also a highly androgenic hormone, when compared with testosterone; its 5 times more androgenic than it.

Testosterone measures at 100 while Trenbolone measures at 500. Potent androgenic anabolics offer dry gains which is great for limiting water retention but don’t think that Tren is water retention free; as it is derived from a progesterone after all. Again as stated due to its great feed efficiency properties, it has potent nutrient partritioning effects. This means more glycogen for the muscle, and less glucose in the adipose tissues. If the compound has higher androgen binding, it will cause the androgen receptors within the fat cells to burn fat more efficiently. This could be alsoe from the rise in leptin with the simulatenous rise in androgens. There is evidence that shows when leptin rises while androgens rise, the body will burn fat at a faster rate to try to lower leptin. One thing to remember with Trenbolone is that it does lower thyroid hormones. This is why you will notice lets of bodybuilders use T3 or T4 while on cycle.

Other perks of Tren are that it does not aromatize into estrogen/estradiol nor does it convert to 5a reductase hormone also known as DHT. The lack of conversions prevents early aggravation of the prostate, estradiol based gynecomastia but that does not mean its gyno free which we will dive into further later.

There are a couple of forms that Trenbolone can be administered in order to gain the benefits of added muscle mass and strength.The two most common forms of Trenbolone are Trenbolone Acetate and Trenobolone Ethanate. The way these esters work is that the plasma lipases cleave its ester group once it reaches the bloodstream leaving free trenbolone. Trenbolone and 17epi-trenbolone are both excreted through urine as conjugates that can be hydrolyzed with beta-glucuronidase. This would imply that trenbolone leaves the body as beta-glucuronides or as sulfates. Clearly the preffered form of Trenbolone is the acetate form as it only has a half of life 3 days meaning that it leaves the body fastest yet mg per mg it’s the strongest. Meaning you need less of it to get the intended benefits yet if side effects become too apparent, it would not take long for the sides to calm down after ceasing use. Most people can be fine after 24 hours of ceasing use, but of course it also depends on dose and duration. Any form of Trenbolone usually needs to be taken every other day to keep plasma blood levels concentrated.

As for dosing Tren Acetate, I recommend to start of with 60-90mg eod and see how it treats you. Honestly there is no point in exceeding 100mgs, you will notice that vets will use 120mgs every other day but its not needed to achieve those massive gains, only a slight difference. 8-12 weeks should be enough to gain mass and strength, Tren is rather toxic, for that reason I don’t recommend that one cycle it for longer periods of time. You can run Trenbolone Ethanate 200mg twice a week.

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